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A novel microRNA miR-MTCO3P38 inhibits malignant progression via STAT3/PTTG1/MYC in hepatocellular carcinoma.

作者信息

Guo Deliang, Gu Yang, Ma Dong, Liu Pengpeng, Chen Baiyang, Liu Zhisu, Wu Ping, Liu Quanyan

机构信息

Department of Hepatobiliary and Pancreatic Surgery, Zhongnan Hospital of Wuhan University, Wuhan, Hubei 430071, PR China.

Department of Hepatobiliary and Pancreatic Surgery, The First People's Hospital of Jingmen, Jingmen, Hubei 448000, PR China.

出版信息

Genes Dis. 2021 Nov 27;9(4):845-848. doi: 10.1016/j.gendis.2021.11.006. eCollection 2022 Jul.

DOI:10.1016/j.gendis.2021.11.006
PMID:35685481
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9170612/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/46a6299a8453/figs6.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/2190a70a36a3/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/e3bc01f43a73/figs1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/9c7d712d8375/figs2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/ef4848244c71/figs3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/1f147723e3bd/figs4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/e325fc514fed/figs5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/46a6299a8453/figs6.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/2190a70a36a3/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/e3bc01f43a73/figs1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/9c7d712d8375/figs2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/ef4848244c71/figs3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/1f147723e3bd/figs4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/e325fc514fed/figs5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0621/9170612/46a6299a8453/figs6.jpg

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Deciphering STAT3 signaling potential in hepatocellular carcinoma: tumorigenesis, treatment resistance, and pharmacological significance.

本文引用的文献

1
PTTG1 is involved in TNF-α-related hepatocellular carcinoma via the induction of c-myc.PTTG1 通过诱导 c-myc 参与 TNF-α 相关的肝癌发生。
Cancer Med. 2019 Sep;8(12):5702-5715. doi: 10.1002/cam4.2473. Epub 2019 Aug 6.
2
Heat Shock Protein 90α-Dependent B-Cell-2-Associated Transcription Factor 1 Promotes Hepatocellular Carcinoma Proliferation by Regulating MYC Proto-Oncogene c-MYC mRNA Stability.热休克蛋白 90α 依赖性 B 细胞相关转录因子 1 通过调节原癌基因 c-MYC mRNA 稳定性促进肝癌增殖。
Hepatology. 2019 Apr;69(4):1564-1581. doi: 10.1002/hep.30172. Epub 2018 Oct 4.
3
Non-coding RNA in hepatocellular carcinoma: Mechanisms, biomarkers and therapeutic targets.
解析 STAT3 信号通路在肝细胞癌中的作用:肿瘤发生、治疗耐药及药理学意义。
Cell Mol Biol Lett. 2023 Apr 21;28(1):33. doi: 10.1186/s11658-023-00438-9.
非编码 RNA 在肝细胞癌中的作用:机制、生物标志物和治疗靶点。
J Hepatol. 2017 Sep;67(3):603-618. doi: 10.1016/j.jhep.2017.04.009. Epub 2017 Apr 22.
4
Targeting blockage of STAT3 inhibits hepatitis B virus-related hepatocellular carcinoma.靶向阻断信号转导和转录激活因子3可抑制乙型肝炎病毒相关的肝细胞癌。
Cancer Biol Ther. 2016 Apr 2;17(4):449-56. doi: 10.1080/15384047.2016.1156257.
5
Cancer statistics in China, 2015.《中国癌症统计数据 2015》
CA Cancer J Clin. 2016 Mar-Apr;66(2):115-32. doi: 10.3322/caac.21338. Epub 2016 Jan 25.