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射频电不对称输送技术对面部损伤的修复作用:两例报告

The Reparative Effects of Radio Electric Asymmetric Conveyer Technology on Facial Injuries: A Report of Two Cases.

作者信息

Fontani Vania, Castagna Alessandro, Rinaldi Salvatore

机构信息

Research Department, Rinaldi Fontani Foundation, Florence, ITA.

Department of Regenerative Medicine, Rinaldi Fontani Institute, Florence, ITA.

出版信息

Cureus. 2022 Jun 24;14(6):e26273. doi: 10.7759/cureus.26273. eCollection 2022 Jun.

DOI:10.7759/cureus.26273
PMID:35754436
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9228334/
Abstract

Facial injuries are often caused by accidental traumas and can be devastating, as their aesthetic outcomes can impact the social relationships and self-esteem of the patient. The reparative processes can be delayed and hindered by the alteration of the endogenous bioelectric activity (EBA) in the damaged tissues, caused by the trauma. In fact, the proper maintenance and generation of EBA is a prerequisite for the cellular health of any tissue and the alteration of EBA determines the inhibition of any cellular repair process, affecting even the cellular differentiation processes. The radio electric asymmetric conveyer (REAC) technology for neurobiological stimulation treatments was designed precisely to restore EBA in both superficial and deep tissues. The two cases of facial trauma presented in this report were treated with the noninvasive treatment of reparative tissue optimization (TO-RPR) applied with REAC technology. The results showed that the REAC TO-RPR treatment can quickly and safely optimize the reparative processes of the tissues, inducing a homogeneous, synchronized, and coordinated recovery, regardless of the type and the aging of the injured tissue and the severity and depth of the lesions.

摘要

面部损伤通常由意外创伤引起,可能具有毁灭性,因为其美学效果会影响患者的社会关系和自尊。创伤会导致受损组织内源性生物电活动(EBA)改变,进而使修复过程延迟并受到阻碍。事实上,EBA的正常维持和产生是任何组织细胞健康的先决条件,EBA的改变会导致任何细胞修复过程受到抑制,甚至影响细胞分化过程。神经生物学刺激治疗的无线电不对称输送器(REAC)技术正是为恢复浅表和深部组织中的EBA而设计的。本报告中呈现的两例面部创伤病例采用了应用REAC技术的非侵入性修复组织优化(TO-RPR)治疗。结果表明,REAC TO-RPR治疗能够快速、安全地优化组织的修复过程,诱导均匀、同步和协调的恢复,而不受受伤组织的类型、老化程度以及损伤的严重程度和深度的影响。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/9d42901e918a/cureus-0014-00000026273-i05.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/e3730c821c51/cureus-0014-00000026273-i01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/46b226ba7522/cureus-0014-00000026273-i02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/645a8ef26c50/cureus-0014-00000026273-i03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/a46fc01d9c32/cureus-0014-00000026273-i04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/9d42901e918a/cureus-0014-00000026273-i05.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/e3730c821c51/cureus-0014-00000026273-i01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/46b226ba7522/cureus-0014-00000026273-i02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/645a8ef26c50/cureus-0014-00000026273-i03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/a46fc01d9c32/cureus-0014-00000026273-i04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4912/9228334/9d42901e918a/cureus-0014-00000026273-i05.jpg

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