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METTL3 promotes proliferation and myogenic differentiation through mA RNA methylation/YTHDF1/2 signaling axis in myoblasts.METTL3 通过 mA RNA 甲基化/YTHDF1/2 信号轴促进成肌细胞增殖和肌生成分化。
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A defined N6-methyladenosine (mA) profile conferred by METTL3 regulates muscle stem cell/myoblast state transitions.由METTL3赋予的特定N6-甲基腺苷（mA）谱调控肌肉干细胞/成肌细胞状态转变。
Cell Death Discov. 2020 Sep 29;6(1):95. doi: 10.1038/s41420-020-00328-5. eCollection 2020.
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Time-course changes in muscle protein degradation in heat-stressed chickens: Possible involvement of corticosterone and mitochondrial reactive oxygen species generation in induction of the ubiquitin-proteasome system.热应激鸡肌肉蛋白质降解的时间进程变化：皮质酮和线粒体活性氧生成可能参与泛素-蛋白酶体系统的诱导
Gen Comp Endocrinol. 2016 Mar 1;228:105-110. doi: 10.1016/j.ygcen.2016.02.007. Epub 2016 Feb 13.
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Pamidronate attenuates muscle loss after pediatric burn injury.帕米膦酸盐可减轻小儿烧伤后肌肉丢失。
J Bone Miner Res. 2014 Jun;29(6):1369-72. doi: 10.1002/jbmr.2162.
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Glucocorticoids and tumor necrosis factor α increase oxidative stress and suppress Wnt protein signaling in osteoblasts.糖皮质激素和肿瘤坏死因子-α增加成骨细胞中的氧化应激并抑制 Wnt 蛋白信号通路。
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Editorial: Osteoporosis and the Role of Muscle.

作者信息

Brotto Marco, Invernizzi Marco, Ireland Alex, Klein Gordon L

机构信息

Director of Bone-Muscle Research Center, University of Texas at Arlington, Arlington, TX, United States.

Department of Health Sciences, University of Eastern Piedmont, Novara, Italy.

出版信息

Front Endocrinol (Lausanne). 2022 Jun 27;13:951298. doi: 10.3389/fendo.2022.951298. eCollection 2022.

DOI:10.3389/fendo.2022.951298
PMID:35832428
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9271953/
Abstract
摘要