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Apoptotic retinal ganglion cell loss is accompanied by complement and cytokine response in the βB1-CTGF primary open-angle glaucoma mouse model.

作者信息

Mueller-Buehl Ana Maria, Reinehr Sabrina

机构信息

Experimental Eye Research Institute, University Eye Hospital, Ruhr-University Bochum, Bochum, Germany.

出版信息

Neural Regen Res. 2023 Feb;18(2):337-338. doi: 10.4103/1673-5374.343911.

DOI:10.4103/1673-5374.343911
PMID:35900424
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9396493/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8902/9396493/57b38ddfcdb7/NRR-18-337-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8902/9396493/57b38ddfcdb7/NRR-18-337-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8902/9396493/57b38ddfcdb7/NRR-18-337-g001.jpg

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Cytokine and Complement Response in the Glaucomatous βB1-CTGF Mouse Model.青光眼βB1-CTGF小鼠模型中的细胞因子和补体反应
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Activation of Apoptosis in a βB1-CTGF Transgenic Mouse Model.βB1-CTGF 转基因小鼠模型中的细胞凋亡激活。
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本文引用的文献

1
Cytokine and Complement Response in the Glaucomatous βB1-CTGF Mouse Model.青光眼βB1-CTGF小鼠模型中的细胞因子和补体反应
Front Cell Neurosci. 2021 Nov 18;15:718087. doi: 10.3389/fncel.2021.718087. eCollection 2021.
2
The Role of Complement in Synaptic Pruning and Neurodegeneration.补体在突触修剪和神经退行性变中的作用。
Immunotargets Ther. 2021 Sep 24;10:373-386. doi: 10.2147/ITT.S305420. eCollection 2021.
3
Activation of Apoptosis in a βB1-CTGF Transgenic Mouse Model.βB1-CTGF 转基因小鼠模型中的细胞凋亡激活。
Int J Mol Sci. 2021 Feb 17;22(4):1997. doi: 10.3390/ijms22041997.
4
Intravitreal Therapy Against the Complement Factor C5 Prevents Retinal Degeneration in an Experimental Autoimmune Glaucoma Model.玻璃体内注射抗补体因子C5治疗可预防实验性自身免疫性青光眼模型中的视网膜变性。
Front Pharmacol. 2019 Dec 2;10:1381. doi: 10.3389/fphar.2019.01381. eCollection 2019.
5
Loss of retinal ganglion cells in a new genetic mouse model for primary open-angle glaucoma.原发性开角型青光眼新型遗传小鼠模型中的视网膜神经节细胞丢失。
J Cell Mol Med. 2019 Aug;23(8):5497-5507. doi: 10.1111/jcmm.14433. Epub 2019 May 29.
6
Methods to Induce Chronic Ocular Hypertension: Reliable Rodent Models as a Platform for Cell Transplantation and Other Therapies.诱导慢性眼高压的方法：作为细胞移植和其他疗法平台的可靠啮齿动物模型。
Cell Transplant. 2018 Feb;27(2):213-229. doi: 10.1177/0963689717724793.
7
Connective tissue growth factor causes glaucoma by modifying the actin cytoskeleton of the trabecular meshwork.结缔组织生长因子通过改变小梁网的肌动蛋白细胞骨架引起青光眼。
Am J Pathol. 2012 Jun;180(6):2386-403. doi: 10.1016/j.ajpath.2012.02.030. Epub 2012 Apr 26.
8
Oxidative stress and the regulation of complement activation in human glaucoma.氧化应激与人类青光眼补体激活的调节
Invest Ophthalmol Vis Sci. 2010 Oct;51(10):5071-82. doi: 10.1167/iovs.10-5289. Epub 2010 May 19.
9
Connective tissue growth factor induces extracellular matrix deposition in human trabecular meshwork cells.结缔组织生长因子诱导人小梁网细胞中的细胞外基质沉积。
Exp Eye Res. 2009 Jun;88(6):1065-75. doi: 10.1016/j.exer.2009.01.008. Epub 2009 Feb 7.
10
The aqueous levels of TGF-beta2 in patients with glaucoma.青光眼患者房水中转化生长因子β2的水平。
Int Ophthalmol. 2004 Jan;25(1):19-22. doi: 10.1023/b:inte.0000018524.48581.79.