脑老化中的生理或病理分子改变。
Physiological or Pathological Molecular Alterations in Brain Aging.
机构信息
Department of Chemical, Biological, Pharmaceutical and Environmental Sciences, University of Messina, 98166 Messina, Italy.
Department of Biomedical, Dental and Morphological and Functional Imaging, University of Messina, 98122 Messina, Italy.
出版信息
Int J Mol Sci. 2022 Aug 2;23(15):8601. doi: 10.3390/ijms23158601.
Abstract
Aging is a natural phenomenon that occurs due to a variety of loosely understood mechanisms [...].
摘要
衰老是一种自然现象，它是由各种机制引起的，这些机制[...]。
相似文献
1
Physiological or Pathological Molecular Alterations in Brain Aging.脑老化中的生理或病理分子改变。
Int J Mol Sci. 2022 Aug 2;23(15):8601. doi: 10.3390/ijms23158601.
2
Nucleus-cytoplasm cross-talk in the aging brain.核质对话在衰老大脑中的作用。
J Neurosci Res. 2020 Feb;98(2):247-261. doi: 10.1002/jnr.24446. Epub 2019 May 26.
3
[Cellular and vascular aging of the brain].[大脑的细胞与血管衰老]
Bull Schweiz Akad Med Wiss. 1980 Apr;36(1-3):57-70.
4
Protein kinase C signal transduction regulation in physiological and pathological aging.生理和病理衰老过程中的蛋白激酶C信号转导调控
Ann N Y Acad Sci. 2005 Dec;1057:177-92. doi: 10.1196/annals.1356.011.
5
The aging brain.衰老的大脑。
Annu Rev Pathol. 2008;3:41-66. doi: 10.1146/annurev.pathmechdis.2.010506.092044.
6
Role of calcium in brain aging.钙在大脑衰老中的作用。
Gen Pharmacol. 1995 Dec;26(8):1651-7. doi: 10.1016/0306-3623(95)00043-7.
7
[Normal and pathological aging of the brain].[大脑的正常与病理性衰老]
Psychiatr Neurol Med Psychol (Leipz). 1955 Jun;7(6):161-74.
8
Acetylome regulation by sirtuins in the brain: from normal physiology to aging and pathology.脑内的 sirtuins 通过乙酰化组学进行调节：从正常生理学到衰老和病理学。
Curr Pharm Des. 2013;19(38):6823-38. doi: 10.2174/1381612811319380014.
9
[Physiological and pathological aging of the brain].[大脑的生理与病理衰老]
Fortschr Med. 1976 May 20;94(15):857-60.
10
Linking pathways in the developing and aging brain with neurodegeneration.将发育中和衰老大脑中的通路与神经退行性变联系起来。
Neuroscience. 2014 Jun 6;269:152-72. doi: 10.1016/j.neuroscience.2014.03.045. Epub 2014 Apr 1.
本文引用的文献
1
Epigenetic Studies in the Male APP/BIN1/COPS5 Triple-Transgenic Mouse Model of Alzheimer's Disease.阿尔茨海默病 APP/BIN1/COPS5 三转基因雄性小鼠模型中的表观遗传学研究。
Int J Mol Sci. 2022 Feb 23;23(5):2446. doi: 10.3390/ijms23052446.
2
Atrazine Inhalation Causes Neuroinflammation, Apoptosis and Accelerating Brain Aging.西玛津吸入会引起神经炎症、细胞凋亡并加速大脑衰老。
Int J Mol Sci. 2021 Jul 26;22(15):7938. doi: 10.3390/ijms22157938.
3
Health Benefits Related to Tree Nut Consumption and Their Bioactive Compounds.坚果摄入与健康益处及其生物活性成分。
Int J Mol Sci. 2021 May 31;22(11):5960. doi: 10.3390/ijms22115960.
4
Selective Activation of CNS and Reference Promoters Is Associated with Distinct Gene Programs Relevant for Neurodegenerative Diseases.选择性激活中枢神经系统和参照启动子与与神经退行性疾病相关的独特基因程序有关。
Int J Mol Sci. 2021 Mar 24;22(7):3296. doi: 10.3390/ijms22073296.
5
Brain alterations in GABA, glutamate and glutamine markers after chronic atrazine exposure in the male albino rat.慢性莠去津暴露后雄性白化大鼠 GABA、谷氨酸和谷氨酰胺标志物的大脑改变。
Arch Toxicol. 2020 Sep;94(9):3217-3230. doi: 10.1007/s00204-020-02806-2. Epub 2020 Jun 19.
6
Adaptation to oxidative stress at cellular and tissue level.细胞和组织水平的氧化应激适应。
Arch Physiol Biochem. 2022 Apr;128(2):521-531. doi: 10.1080/13813455.2019.1702059. Epub 2019 Dec 13.
7
Ageing, age-related diseases and oxidative stress: What to do next?衰老、与年龄相关的疾病和氧化应激：下一步该做什么？
Ageing Res Rev. 2020 Jan;57:100982. doi: 10.1016/j.arr.2019.100982. Epub 2019 Nov 13.
8
Gene-based analysis in HRC imputed genome wide association data identifies three novel genes for Alzheimer's disease.基于基因的分析在 HRC 导入基因组全基因组关联数据中确定了三个新的阿尔茨海默病基因。
PLoS One. 2019 Jul 8;14(7):e0218111. doi: 10.1371/journal.pone.0218111. eCollection 2019.
9
Mitophagy in neurodegeneration and aging.神经退行性疾病和衰老中的自噬。
Neurochem Int. 2017 Oct;109:202-209. doi: 10.1016/j.neuint.2017.02.007. Epub 2017 Feb 21.
10
PGC-1α is a male-specific disease modifier of human and experimental amyotrophic lateral sclerosis.PGC-1α 是人类和实验性肌萎缩侧索硬化症的男性特异性疾病修饰因子。
Hum Mol Genet. 2013 Sep 1;22(17):3477-84. doi: 10.1093/hmg/ddt202. Epub 2013 May 12.
Zotero 插件