Bothor C, Berg W, Pirlich W
Urologe A. 1987 May;26(3):133-6.
Acidosis induced increase in renal calcium excretion was produced by means of ammonium chloride load in 12 control persons and in 76 patients with recurrent oxalate lithiasis. This increase is more marked in stone patients - even in those with normocalciuria - than in control persons. The calcium excretion is especially increased in patients with renal hypercalciuria. This might result from the primary nature of the renal defect. The acid load did not prove useful in the diagnosis of hyperparathyroidism.
通过氯化铵负荷试验,在12名对照者和76例复发性草酸钙结石患者中诱发了酸中毒所致的肾钙排泄增加。这种增加在结石患者中更为明显——即使是那些正常钙尿患者——比对照者更明显。肾高钙尿患者的钙排泄尤其增加。这可能是由于肾脏缺陷的原发性所致。酸负荷试验在甲状旁腺功能亢进的诊断中并无用处。