• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Correction to: Propionibacterium acnes overabundance in gastric cancer promote M2 polarization of macrophages via a TLR4/PI3K/Akt signaling.

作者信息

Li Qing, Wu Wei, Gong Dexin, Shang Renduo, Wang Jing, Yu Honggang

机构信息

Department of Gastroenterology, Renmin Hospital of Wuhan University, Wuhan, 430060, People's Republic of China.

Hubei Key Laboratory of Digestive System Disease, Renmin Hospital of Wuhan University, Wuhan, 430060, People's Republic of China.

出版信息

Gastric Cancer. 2022 Nov;25(6):1139-1141. doi: 10.1007/s10120-022-01339-0.

DOI:10.1007/s10120-022-01339-0
PMID:36087162
Abstract
摘要

相似文献

1
Correction to: Propionibacterium acnes overabundance in gastric cancer promote M2 polarization of macrophages via a TLR4/PI3K/Akt signaling.对以下内容的修正:胃癌中痤疮丙酸杆菌的过度生长通过TLR4/PI3K/Akt信号通路促进巨噬细胞的M2极化。
Gastric Cancer. 2022 Nov;25(6):1139-1141. doi: 10.1007/s10120-022-01339-0.
2
Propionibacterium acnes overabundance in gastric cancer promote M2 polarization of macrophages via a TLR4/PI3K/Akt signaling.痤疮丙酸杆菌在胃癌中的过度存在通过TLR4/PI3K/Akt信号通路促进巨噬细胞的M2极化。
Gastric Cancer. 2021 Nov;24(6):1242-1253. doi: 10.1007/s10120-021-01202-8. Epub 2021 Jun 2.
3
Crosstalk between tumor-associated macrophages and tumor cells promotes chemoresistance via CXCL5/PI3K/AKT/mTOR pathway in gastric cancer.肿瘤相关巨噬细胞与肿瘤细胞之间的串扰通过CXCL5/PI3K/AKT/mTOR途径促进胃癌的化疗耐药性。
Cancer Cell Int. 2022 Sep 23;22(1):290. doi: 10.1186/s12935-022-02717-5.
4
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.SMAD-PI3K-Akt-mTOR信号通路介导骨形态发生蛋白7将单核细胞极化成为M2巨噬细胞。
PLoS One. 2013 Dec 20;8(12):e84009. doi: 10.1371/journal.pone.0084009. eCollection 2013.
5
[Gastric cancer-derived mesenchymal stem cells regulate the M2 polarization of macrophages within gastric cancer microenvironment via JAK2/STAT3 signaling pathway].胃癌来源的间充质干细胞通过JAK2/STAT3信号通路调节胃癌微环境中巨噬细胞的M2极化
Zhonghua Zhong Liu Za Zhi. 2022 Jul 23;44(7):728-736. doi: 10.3760/cma.j.cn112152-20200106-00008.
6
Selective priming to Toll-like receptor 4 (TLR4), not TLR2, ligands by P. acnes involves up-regulation of MD-2 in mice.痤疮丙酸杆菌对Toll样受体4(TLR4)而非TLR2配体的选择性启动涉及小鼠中MD-2的上调。
Hepatology. 2004 Sep;40(3):555-64. doi: 10.1002/hep.20350.
7
Macrophage migration inhibitory factor of Thelazia callipaeda induces M2-like macrophage polarization through TLR4-mediated activation of the PI3K-Akt pathway.犬首线虫巨噬细胞移动抑制因子通过 TLR4 介导的 PI3K-Akt 通路激活诱导 M2 样巨噬细胞极化。
FASEB J. 2021 Sep;35(9):e21866. doi: 10.1096/fj.202100676R.
8
Propionibacterium acnes induces an adjuvant effect in B-1 cells and affects their phagocyte differentiation via a TLR2-mediated mechanism.痤疮丙酸杆菌在B-1细胞中诱导佐剂效应,并通过Toll样受体2(TLR2)介导的机制影响其吞噬细胞分化。
Immunobiology. 2016 Sep;221(9):1001-11. doi: 10.1016/j.imbio.2016.03.003. Epub 2016 May 7.
9
SPARC induces M2 polarization of macrophages to promote proliferation, migration, and angiogenesis of cholangiocarcinoma cells.基质细胞关联基因 2(SPARC)可诱导巨噬细胞向 M2 型极化,从而促进胆管癌细胞的增殖、迁移和血管生成。
Neoplasma. 2022 Sep;69(5):1101-1107. doi: 10.4149/neo_2022_220324N333. Epub 2022 Aug 12.
10
Propionibacterium acnes overabundance and natural killer group 2 member D system activation in corpus-dominant lymphocytic gastritis.痤疮丙酸杆菌过度增殖与自然杀伤细胞2型成员D系统在以淋巴细胞为主的胃体胃炎中的激活
J Pathol. 2016 Dec;240(4):425-436. doi: 10.1002/path.4782. Epub 2016 Oct 21.