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Editorial: Cellular and molecular factors that drive the behavior of oligodendrocyte progenitor cells in physiological conditions and disease.

作者信息

Lecca Davide, Baron Wia, Butt Arthur M

机构信息

Department of Pharmaceutical Sciences, Università degli Studi di Milano, Milan, Italy.

Department of Biomedical Sciences of Cells and Systems, University of Groningen, University Medical Center Groningen, Groningen, Netherlands.

出版信息

Front Cell Neurosci. 2023 Feb 6;17:1145627. doi: 10.3389/fncel.2023.1145627. eCollection 2023.

DOI:10.3389/fncel.2023.1145627
PMID:36814864
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9939830/
Abstract
摘要

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Cells. 2022 Aug 2;11(15):2369. doi: 10.3390/cells11152369.
2
Molecular and functional heterogeneity in dorsal and ventral oligodendrocyte progenitor cells of the mouse forebrain in response to DNA damage.在应对 DNA 损伤时，小鼠前脑背侧和腹侧少突胶质前体细胞的分子和功能异质性。
Nat Commun. 2022 Apr 28;13(1):2331. doi: 10.1038/s41467-022-30010-6.
3
Functional genomic analyses highlight a shift in Gpr17-regulated cellular processes in oligodendrocyte progenitor cells and underlying myelin dysregulation in the aged mouse cerebrum.功能基因组分析突出了在少突胶质前体细胞中 Gpr17 调节的细胞过程的转变以及衰老小鼠大脑中髓鞘失调的潜在机制。
Aging Cell. 2021 Apr;20(4):e13335. doi: 10.1111/acel.13335. Epub 2021 Mar 5.
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Functionally distinct subgroups of oligodendrocyte precursor cells integrate neural activity and execute myelin formation.功能不同的少突胶质前体细胞亚群整合神经活动并执行髓鞘形成。
Nat Neurosci. 2020 Mar;23(3):363-374. doi: 10.1038/s41593-019-0581-2. Epub 2020 Feb 17.
5
Cellular senescence in progenitor cells contributes to diminished remyelination potential in progressive multiple sclerosis.祖细胞中的细胞衰老导致进行性多发性硬化症中髓鞘再生潜能降低。
Proc Natl Acad Sci U S A. 2019 Apr 30;116(18):9030-9039. doi: 10.1073/pnas.1818348116. Epub 2019 Mar 25.
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Differential local tissue permissiveness influences the final fate of GPR17-expressing oligodendrocyte precursors in two distinct models of demyelination.差异的局部组织允许性影响 GPR17 表达的少突胶质前体细胞在两种不同脱髓鞘模型中的最终命运。
Glia. 2018 May;66(5):1118-1130. doi: 10.1002/glia.23305. Epub 2018 Feb 9.
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Down-regulation of the mitochondrial aspartate-glutamate carrier isoform 1 AGC1 inhibits proliferation and N-acetylaspartate synthesis in Neuro2A cells.线粒体天冬氨酸-谷氨酸载体同工型 1 (AGC1) 的下调抑制 Neuro2A 细胞的增殖和 N-乙酰天冬氨酸的合成。
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