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骨发育与再生 2.0.

Bone Development and Regeneration 2.0.

机构信息

Department of Frontier Medicine, Institute of Medical Science, St. Marianna University School of Medicine, Kawasaki 216-8511, Japan.

出版信息

Int J Mol Sci. 2023 May 15;24(10):8761. doi: 10.3390/ijms24108761.

DOI:10.3390/ijms24108761
PMID:37240107
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10218555/
Abstract

Bone is an important tissue which is a structural body component, carrying out the roles of mechanical stress response and organ/tissue protection [...].

摘要

骨骼是一种重要的组织，是身体结构的组成部分，具有机械应力响应和器官/组织保护的作用[...]。

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本文引用的文献

[1]
Mechanisms during Osteogenic Differentiation in Human Dental Follicle Cells.

Int J Mol Sci. 2022-5-25

[2]
Transcription Factor Lmx1b Negatively Regulates Osteoblast Differentiation and Bone Formation.

Int J Mol Sci. 2022-5-7

[3]
, , and Are the Most Stably Expressed Genes during Osteogenic Differentiation.

Int J Mol Sci. 2022-4-12

[4]
Cytoskeletal Protein 4.1G Is Essential for the Primary Ciliogenesis and Osteoblast Differentiation in Bone Formation.

Int J Mol Sci. 2022-2-14

[5]
Physiologic Mechanical Stress Directly Induces Bone Formation by Activating Glucose Transporter 1 (Glut 1) in Osteoblasts, Inducing Signaling via NAD+-Dependent Deacetylase (Sirtuin 1) and Runt-Related Transcription Factor 2 (Runx2).

Int J Mol Sci. 2021-8-23

[6]
In Vivo Osteocyte Mechanotransduction: Recent Developments and Future Directions.

Curr Osteoporos Rep. 2018-12

[7]
Glucose dependent miR-451a expression contributes to parathyroid hormone mediated osteoblast differentiation.

Bone. 2018-9-13

[8]
Mst1/2 Kinases Modulate Glucose Uptake for Osteoblast Differentiation and Bone Formation.

J Bone Miner Res. 2018-3-24

[9]
Vhl deletion in osteoblasts boosts cellular glycolysis and improves global glucose metabolism.

J Clin Invest. 2018-2-12

[10]
Osteocyte Mechanobiology.

Curr Osteoporos Rep. 2017-8

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