粗糙脉孢菌中硝酸还原酶的氮代谢物阻遏：gln-1a基因座的作用。
Nitrogen metabolite repression of nitrate reductase in Neurospora crassa: effect of the gln-1a locus.
作者信息
Dunn-Coleman N S, Tomsett A B, Garrett R H
出版信息
J Bacteriol. 1979 Aug;139(2):697-700. doi: 10.1128/jb.139.2.697-700.1979.
Abstract
Nicotinamide adenine dinucleotide phosphate (reduced form)-nitrate reductase was freed from ammonium repression in a Neurospora crassa mutant having drastically lowered glutamine synthetase activity, gln-1a. The general phenomenon of nitrogen metabolite repression required glutamine or some aspect of glutamine metabolism.
摘要
烟酰胺腺嘌呤二核苷酸磷酸（还原型）-硝酸还原酶在谷氨酰胺合成酶活性大幅降低的粗糙脉孢菌突变体gln-1a中摆脱了铵阻遏。氮代谢物阻遏的普遍现象需要谷氨酰胺或谷氨酰胺代谢的某些方面。
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