Department of Psychology, Northwestern University.
Department of Psychology, University of California, Los Angeles.
J Psychopathol Clin Sci. 2023 Aug;132(6):645-656. doi: 10.1037/abn0000803. Epub 2023 Jun 1.
This study aimed to characterize within-person pre-COVID-19 and coronavirus pandemic (COVID-19) transdiagnostic anxiety and depression symptom trajectories in emerging adults and determine the roles of neuroticism and behavioral activation in predicting these COVID-19-related changes. We recruited a sample of 342 emerging adults (aged 18-19 at baseline) who were screened on neuroticism and behavioral activation and completed symptom questionnaires on multiple occasions before and after the start of the pandemic. We examined estimates of the symptom factors of General Distress, Anhedonia-Apprehension, and Fears at each wave. The stress amplification model predicts a multiplicative neuroticism-adversity interaction with those high on neuroticism showing the greatest symptom increases to the pandemic. The stably elevated negative affect model is an additive model and predicts that persons high on neuroticism will display elevated symptoms at every wave. General Distress and Anhedonia-Apprehension showed large increases from the pre-COVID-19 to COVID-19 transition then decreased thereafter. The increase brought the average General Distress score to clinical levels at the first COVID-19 wave. There was a small decrease in Fears from the pre-COVID-19 to COVID-19 transition followed by a large increase. Thus, COVID-19 was associated with both increases in psychological symptoms and some resilience. Neuroticism positively predicted the pre-COVID-19 to COVID-19 transition change in Fears but was associated with a of increases in General Distress and Anhedonia-Apprehension. The results disconfirmed the stress amplification model of neuroticism but partially supported the stably elevated negative affect model. (PsycInfo Database Record (c) 2023 APA, all rights reserved).
本研究旨在描述新冠疫情前和疫情期间(COVID-19)成年早期个体的跨诊断焦虑和抑郁症状轨迹,并确定神经质和行为激活在预测这些与 COVID-19 相关变化中的作用。我们招募了 342 名成年早期个体(基线时年龄为 18-19 岁),他们在神经质和行为激活方面接受了筛选,并在疫情开始前后多次完成症状问卷。我们在每个波次检查了一般困扰、快感缺乏-焦虑和恐惧的症状因子的估计值。应激放大模型预测了神经质与逆境的乘法交互作用,即神经质水平高的人对疫情的症状增加最大。稳定升高的负性情绪模型是一个加法模型,预测神经质水平高的人在每个波次都会表现出升高的症状。一般困扰和快感缺乏-焦虑在从 COVID-19 前到 COVID-19 的转变中出现了大幅增加,然后在此后下降。这种增加使平均一般困扰得分在第一次 COVID-19 波次达到临床水平。恐惧在从 COVID-19 前到 COVID-19 的转变中略有下降,随后大幅增加。因此,COVID-19 既与心理症状的增加有关,也与一些弹性有关。神经质正向预测了从 COVID-19 前到 COVID-19 的转变中恐惧的变化,但与一般困扰和快感缺乏-焦虑的增加有关。结果否定了神经质的应激放大模型,但部分支持了稳定升高的负性情绪模型。