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烟草中PR(b)蛋白的化学诱导及对烟草花叶病毒感染的抗性

The chemical induction of PR (b) proteins and resistance to TMV infection in tobacco.

作者信息

White R F, Dumas E, Shaw P, Antoniw J F

出版信息

Antiviral Res. 1986 May;6(3):177-85. doi: 10.1016/0166-3542(86)90012-4.

Abstract

Aspirin injected into Xanthi-nc tobacco leaves induces the production of PR protein and resistance to TMV. The concentration of PR protein and resistance increases with increasing aspirin concentration, up to a plateau. 2-Thiouracil and dioxohexahydrotriazine also induce PR protein when injected into tobacco leaves. Barium and manganese salts induced PR protein, but those of eight other cations did not. Certain salts were phytotoxic but did not induce PR protein, confirming that the production of PR protein is not a non-specific stress response.

摘要

将阿司匹林注射到黄花烟草叶片中可诱导病程相关蛋白(PR蛋白)的产生并增强对烟草花叶病毒(TMV)的抗性。PR蛋白的浓度和抗性随阿司匹林浓度的增加而升高,直至达到平稳状态。将2-硫尿嘧啶和二氧六氢三嗪注射到烟草叶片中也可诱导PR蛋白的产生。钡盐和锰盐可诱导PR蛋白的产生,但其他八种阳离子的盐类则不能。某些盐类具有植物毒性,但不能诱导PR蛋白的产生,这证实了PR蛋白的产生并非非特异性应激反应。

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