de Leeuw P W, Waltman F L, Birkenhäger W H
Hypertension. 1986 Sep;8(9):810-2. doi: 10.1161/01.hyp.8.9.810.
A 40-year-old man was admitted to the hospital with pulmonary edema without signs of left ventricular failure. Noncardiogenic pulmonary edema was diagnosed, and a subsequent workup identified a pheochromocytoma as the cause of this condition. The clinical picture could be mimicked by infusion of exogenous norepinephrine. It is concluded that surges of catecholamines from a pheochromocytoma may provoke pulmonary edema in a manner similar to that by which neurogenic pulmonary edema related to cerebral disorders occurs.
一名40岁男性因肺水肿入院,无左心室衰竭迹象。诊断为非心源性肺水肿,随后的检查发现嗜铬细胞瘤是导致该病症的原因。外源性去甲肾上腺素输注可模拟该临床症状。得出的结论是,嗜铬细胞瘤释放的儿茶酚胺激增可能以类似于与脑部疾病相关的神经源性肺水肿的发生方式引发肺水肿。