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类风湿关节炎发病机制的分子机制 1 和 2 的突破。

The Pathogenesis of Rheumatoid Arthritis Breakthroughs in Molecular Mechanisms 1 and 2.

机构信息

Institute of Rheumatology, Tokyo Women's Medical University, Tokyo 162-8666, Japan.

Division of Rheumatology, First Department of Comprehensive Medicine, Jichi Medical University Saitama Medical Center, Saitama 330-0834, Japan.

出版信息

Int J Mol Sci. 2023 Jul 4;24(13):11060. doi: 10.3390/ijms241311060.

DOI:10.3390/ijms241311060
PMID:37446237
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10341906/
Abstract

The pathogenesis of rheumatoid arthritis (RA) consists of the formation of synovial villi, inflammation, immune abnormalities, and bone-cartilage destruction [...].

摘要

类风湿关节炎(RA)的发病机制包括滑膜绒毛的形成、炎症、免疫异常和骨软骨破坏[...]。

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Serum Levels of IFABP2 and Differences in and Abundance on Gut Microbiota Are Associated with Poor Therapeutic Response in Rheumatoid Arthritis: A Pilot Study.血清 IFABP2 水平和肠道微生物群丰度的差异与类风湿关节炎治疗反应不良相关:一项初步研究。
Int J Mol Sci. 2023 Jan 19;24(3):1958. doi: 10.3390/ijms24031958.
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Int J Mol Sci. 2022 Oct 20;23(20):12599. doi: 10.3390/ijms232012599.
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