Quinn Meghan E, Shields Grant S
Department of Psychological Sciences, William & Mary.
Department of Psychological Science, University of Arkansas.
Clin Psychol Sci. 2023 Sep;11(5):773-800. doi: 10.1177/21677026221149736. Epub 2023 Feb 22.
Although exposure to acute stress undoubtedly contributes to psychopathology, most individuals do not develop psychopathology following stress exposure. To explain this, biological, emotional, and cognitive responses to stress have been implicated, but individual differences in executive control (i.e., top-down control of cognition and behavior) measured in response to stress has only recently emerged as a potential factor contributing to psychopathology. In this review, we introduce a model-the -positing how the impairing effects of acute stress on executive control can contribute to psychopathology. We link to research on biological, emotional, and cognitive processes, all of which can be impacted by executive control, to propose a framework for how poorer executive control under conditions of acute stress can contribute to psychopathology. This integrated model is intended to further our understanding of who is more susceptible to the negative consequences of stress.
尽管暴露于急性应激无疑会导致精神病理学,但大多数人在经历应激后并不会患上精神病理学。为了解释这一点,人们已经将对应激的生物、情感和认知反应牵连其中,但直到最近,在应激反应中测量的执行控制(即对认知和行为的自上而下的控制)方面的个体差异才作为导致精神病理学的一个潜在因素出现。在这篇综述中,我们介绍了一个模型——假定急性应激对执行控制的损害作用如何导致精神病理学。我们联系了关于生物、情感和认知过程的研究,所有这些过程都可能受到执行控制的影响,以提出一个框架,说明在急性应激条件下较差的执行控制如何导致精神病理学。这个综合模型旨在进一步加深我们对谁更容易受到应激负面影响的理解。