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Editorial: Oxidative stress link associated with mitochondrial bioenergetics: relevance in cell aging and age-related pathologies.

作者信息

Kalra Rajkumar Singh, Kumar Subodh, Tomar Dhanendra

机构信息

Immune Signal Unit, Okinawa Institute of Science and Technology Graduate University, Okinawa, Japan.

Department of Molecular and Translational Medicine, Paul L. Foster School of Medicine, Texas Tech University Health Sciences Center El Paso, El Paso, TX, United States.

出版信息

Front Cell Dev Biol. 2023 Sep 5;11:1273420. doi: 10.3389/fcell.2023.1273420. eCollection 2023.

DOI:10.3389/fcell.2023.1273420
PMID:37736499
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10509040/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d70e/10509040/651080e00881/fcell-11-1273420-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d70e/10509040/651080e00881/fcell-11-1273420-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d70e/10509040/651080e00881/fcell-11-1273420-g001.jpg

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Correction: Inhibition of protein tyrosine phosphatase improves mitochondrial bioenergetics and dynamics, reduces oxidative stress, and enhances adipogenic differentiation potential in metabolically impaired progenitor stem cells.更正：抑制蛋白酪氨酸磷酸酶可改善线粒体生物能量学和动力学，降低氧化应激，并增强代谢受损祖干细胞的脂肪生成分化潜能。
Cell Commun Signal. 2023 Jan 20;21(1):12. doi: 10.1186/s12964-023-01055-x.
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Altered glucose metabolism in Alzheimer's disease: Role of mitochondrial dysfunction and oxidative stress.阿尔茨海默病中葡萄糖代谢的改变：线粒体功能障碍和氧化应激的作用。
Free Radic Biol Med. 2022 Nov 20;193(Pt 1):134-157. doi: 10.1016/j.freeradbiomed.2022.09.032. Epub 2022 Oct 4.
3
Mitochondrial protein dysfunction in pathogenesis of neurological diseases.
线粒体蛋白功能障碍在神经疾病发病机制中的作用
Front Mol Neurosci. 2022 Sep 7;15:974480. doi: 10.3389/fnmol.2022.974480. eCollection 2022.
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Exp Cell Res. 2020 Oct 15;395(2):112240. doi: 10.1016/j.yexcr.2020.112240. Epub 2020 Aug 20.
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