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C/EBP homogenous protein-induced Apoptosis in Endoplasmic Reticulum stress has been implicated in Kikuchi-Fujimoto Disease.

作者信息

Asano Shigeyuki, Yamazaki Kazuki, Mori Kikuo, Hashimoto Yuko, Kawana Satoshi, Sato Hiroko, Naito Hiroyuki, Shikano Koji, Sogame Yoichiro, Kashimura Makoto

机构信息

Pathology Center, Iwaki City Medical Center, Iwaki, Fukushima, Japan.

Department of Diagnostic Pathology, Fukushima Medical University, School of Medicine, Fukushima, Fukushima, Japan.

出版信息

J Clin Exp Hematop. 2023 Dec 26;63(4):270-274. doi: 10.3960/jslrt.23034. Epub 2023 Oct 28.

DOI:10.3960/jslrt.23034
PMID:37899238
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10861369/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/8223776358c5/jslrt-63-270-gK-M.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/2510a2b15fd0/jslrt-63-270-gA-F.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/7b9afea0c8e9/jslrt-63-270-gG-H.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/4298730090b8/jslrt-63-270-gI.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/89b07e2cce29/jslrt-63-270-gJ.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/8223776358c5/jslrt-63-270-gK-M.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/2510a2b15fd0/jslrt-63-270-gA-F.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/7b9afea0c8e9/jslrt-63-270-gG-H.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/4298730090b8/jslrt-63-270-gI.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/89b07e2cce29/jslrt-63-270-gJ.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b60e/10861369/8223776358c5/jslrt-63-270-gK-M.jpg

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1
C/EBP homogenous protein-induced Apoptosis in Endoplasmic Reticulum stress has been implicated in Kikuchi-Fujimoto Disease.C/EBP同源蛋白在内质网应激诱导的细胞凋亡与菊池-藤本病有关。
J Clin Exp Hematop. 2023 Dec 26;63(4):270-274. doi: 10.3960/jslrt.23034. Epub 2023 Oct 28.
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[Kikuchi-Fujimoto disease: about a case].[菊池-藤本病：关于一例病例]
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Histiocytic necrotizing lymphadenitis (Kikuchi-Fujimoto disease) diagnosed by fine needle aspiration biopsy.经细针穿刺活检诊断的组织细胞坏死性淋巴结炎（菊池-藤本病）
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本文引用的文献

1
Kikuchi-Fujimoto disease is mediated by an aberrant type I interferon response.奇库蒂-富古imoto 病是由异常的 I 型干扰素反应介导的。
Mod Pathol. 2022 Apr;35(4):462-469. doi: 10.1038/s41379-021-00992-7. Epub 2021 Dec 24.
2
IFN-γ-induced ER stress impairs autophagy and triggers apoptosis in lung cancer cells.IFN-γ 诱导的内质网应激会损害肺癌细胞中的自噬并引发细胞凋亡。
Oncoimmunology. 2021 Aug 10;10(1):1962591. doi: 10.1080/2162402X.2021.1962591. eCollection 2021.
3
Necrotizing lymphadenitis may be induced by overexpression of Toll-like receptor7 (TLR7) caused by reduced TLR9 transport in plasmacytoid dendritic cells (PDCs).
浆细胞样树突状细胞（PDCs）中 TLR9 转运减少导致 Toll 样受体 7（TLR7）过度表达可能引发坏死性淋巴结炎。
J Clin Exp Hematop. 2021 Jun 5;61(2):85-92. doi: 10.3960/jslrt.20060. Epub 2021 May 14.
4
Redox-assisted regulation of Ca2+ homeostasis in the endoplasmic reticulum by disulfide reductase ERdj5.二硫键还原酶ERdj5对内质网中Ca2+稳态的氧化还原辅助调节
Proc Natl Acad Sci U S A. 2016 Oct 11;113(41):E6055-E6063. doi: 10.1073/pnas.1605818113. Epub 2016 Sep 30.
5
Plasmacytoid Dendritic Cells Producing Interferon-α (IFN-α) and Inducing Mx1 Play an Important Role for CD4(+) Cells and CD8(+) Cells in Necrotizing Lymphadenitis.产生干扰素-α（IFN-α）并诱导Mx1的浆细胞样树突状细胞在坏死性淋巴结炎中对CD4(+)细胞和CD8(+)细胞起重要作用。
J Clin Exp Hematop. 2015;55(3):127-35. doi: 10.3960/jslrt.55.127.
6
Transient Ca2+ depletion from the endoplasmic reticulum is critical for skeletal myoblast differentiation.内质网中短暂的钙离子耗竭对骨骼肌成肌细胞分化至关重要。
FASEB J. 2015 May;29(5):2137-49. doi: 10.1096/fj.14-261529. Epub 2015 Feb 12.
7
New insights into the roles of CHOP-induced apoptosis in ER stress.CHOP诱导的细胞凋亡在内质网应激中的作用的新见解。
Acta Biochim Biophys Sin (Shanghai). 2014 Aug;46(8):629-40. doi: 10.1093/abbs/gmu048.
8
Necrotizing lymphadenitis (NEL) is a systemic disease characterized by blastic transformation of CD8+ cells and apoptosis of CD4+ cells.坏死性淋巴结炎（NEL）是一种全身性疾病，其特征为 CD8+细胞的母细胞转化和 CD4+细胞的凋亡。
Virchows Arch. 2014 Jan;464(1):95-103. doi: 10.1007/s00428-013-1516-z. Epub 2013 Nov 30.
9
Arsenic induces reactive oxygen species-caused neuronal cell apoptosis through JNK/ERK-mediated mitochondria-dependent and GRP 78/CHOP-regulated pathways.砷通过 JNK/ERK 介导的线粒体依赖性和 GRP 78/CHOP 调节的途径诱导活性氧引起的神经元细胞凋亡。
Toxicol Lett. 2014 Jan 3;224(1):130-40. doi: 10.1016/j.toxlet.2013.10.013. Epub 2013 Oct 21.
10
The eIF2 kinase PERK and the integrated stress response facilitate activation of ATF6 during endoplasmic reticulum stress.内质网应激过程中，真核起始因子 2 激酶 PERK 和综合应激反应促进 ATF6 的激活。
Mol Biol Cell. 2011 Nov;22(22):4390-405. doi: 10.1091/mbc.E11-06-0510. Epub 2011 Sep 14.