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本文引用的文献

1
Mitochondrial dysfunction in human hypertrophic cardiomyopathy is linked to cardiomyocyte architecture disruption and corrected by improving NADH-driven mitochondrial respiration.人类肥厚型心肌病中线粒体功能障碍与心肌细胞结构破坏有关，并通过改善 NADH 驱动的线粒体呼吸得到纠正。
Eur Heart J. 2023 Apr 1;44(13):1170-1185. doi: 10.1093/eurheartj/ehad028.
2
Loss of Nuclear Envelope Integrity and Increased Oxidant Production Cause DNA Damage in Adult Hearts Deficient in PKP2: A Molecular Substrate of ARVC.PKP2 缺乏导致成年心脏核膜完整性丧失和活性氧产生增加，从而引起 DNA 损伤：ARVC 的分子底物。
Circulation. 2022 Sep 13;146(11):851-867. doi: 10.1161/CIRCULATIONAHA.122.060454. Epub 2022 Aug 12.
3
SR-Mitochondria Crosstalk Shapes Ca Signalling to Impact Pathophenotype in Disease Models Marked by Dysregulated Intracellular Ca Release.SR-线粒体串扰影响钙信号转导，从而影响以细胞内钙释放失调为特征的疾病模型中的表型。
Cardiovasc Res. 2022 Oct 21;118(13):2819-2832. doi: 10.1093/cvr/cvab324.
4
Loss of Mitochondrial Ca Uniporter Limits Inotropic Reserve and Provides Trigger and Substrate for Arrhythmias in Barth Syndrome Cardiomyopathy.线粒体钙单向转运体缺失限制了心肌变力储备，并为巴特综合征心肌病心律失常提供触发和底物。
Circulation. 2021 Nov 23;144(21):1694-1713. doi: 10.1161/CIRCULATIONAHA.121.053755. Epub 2021 Oct 14.

Top Stories: Mitochondrial origin of inherited cardiac arrhythmias.

作者信息

Akar Fadi G, Maack Christoph

机构信息

Department of Internal Medicine, Section of Cardiovascular Medicine, Yale School of Medicine, New Haven, Connecticut; Department of Biomedical Engineering, Yale University School of Engineering & Applied Sciences, New Haven, Connecticut.

Department of Translational Research, Comprehensive Heart Failure Center (CHFC), University Clinic Würzburg, Würzburg, Germany.

出版信息

Heart Rhythm. 2024 Feb;21(2):235-236. doi: 10.1016/j.hrthm.2023.10.020.

DOI:10.1016/j.hrthm.2023.10.020
PMID:38296456
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10857749/
Abstract
摘要