Beel Wouter, Macchiarola Luca, Mouton Caroline, Laver Lior, Seil Romain
Department of Orthopaedic Surgery, Centre Hospitalier de Luxembourg - Clinique d'Eich, Luxembourg, Luxembourg.
Department of Orthopaedics and Traumatology, Cantonal Hospital Winterthur, Zurich, Switzerland.
Ann Jt. 2022 Apr 15;7:14. doi: 10.21037/aoj-21-9. eCollection 2022.
The purpose of this review is to improve the awareness of lateral meniscal hypermobility by describing its relevant anatomy, biomechanics, pathophysiology, imaging and arthroscopic findings as well as the treatment options.
The lateral meniscus is less stable than the medial meniscus. Its important posterior stabilizers are the popliteomeniscal fascicles, the posterior capsule, the meniscofemoral ligaments and the posterior meniscotibial ligament, which are divided by a bare area, the popliteal hiatus. Atraumatic insufficiency or rupture of one of these key structures may impact the mobility of the lateral meniscus and can lead to an unstable, hypermobile lateral meniscus. Lateral meniscus hypermobility can cause lateral knee pain and mechanical symptoms as locking. Ruptures of the popliteomeniscal fascicles are frequently associated with anterior cruciate ligament and posterolateral corner injuries. Their repair may be important to fully restore knee stability.
This is a narrative overview of the literature synthesizing current knowledge about the hypermobile lateral meniscus. Anatomy, biomechanics, diagnosis and treatment of this entity was of particular interest for this review. Literature was retrieved from PubMed database, hand searches and cross-reference checking.
Diagnosing lateral meniscus hypermobility is challenging since the magnetic resonance imaging are often unspecific and may show no structural alterations of the meniscus and its attachments. The only hint can be the patient's history and clinical symptoms (e.g., locking). Ultimately, the diagnosis is confirmed during knee arthroscopy, when the lateral meniscus can be mobilized over 50% of the lateral tibial plateau or lateral femoral condyle while anterior probing or by using the aspiration function during arthroscopy. Treatment includes stabilizing the posterior lateral meniscus by repairing the injured structures. Therefore, a systematic arthroscopic evaluation of the lateral compartment is important to fully recognize the problem. Repair can be achieved arthroscopically with various suturing techniques used for meniscal repair with satisfactory results and low recurrence rates.
本综述的目的是通过描述外侧半月板活动度过高的相关解剖结构、生物力学、病理生理学、影像学和关节镜检查结果以及治疗选择,提高对其的认识。
外侧半月板比内侧半月板稳定性差。其重要的后稳定结构是腘半月板束、后关节囊、半月板股骨韧带和后半月板胫骨韧带,它们被一个无血管区即腘裂孔分开。这些关键结构中的任何一个发生非创伤性功能不全或断裂,都可能影响外侧半月板的活动度,导致外侧半月板不稳定、活动度过高。外侧半月板活动度过高可引起膝关节外侧疼痛和交锁等机械性症状。腘半月板束断裂常与前交叉韧带和后外侧角损伤相关。修复这些结构对于完全恢复膝关节稳定性可能很重要。
这是一篇叙述性综述,综合了有关活动度过高的外侧半月板的现有知识。本综述特别关注该疾病的解剖结构、生物力学、诊断和治疗。通过检索PubMed数据库、手工检索和交叉参考文献核对获取文献。
诊断外侧半月板活动度过高具有挑战性,因为磁共振成像通常不具有特异性,可能未显示半月板及其附着结构的结构改变。唯一的线索可能是患者的病史和临床症状(如交锁)。最终,在膝关节镜检查时,当外侧半月板在前探或使用关节镜吸引功能时可在外侧胫骨平台或外侧股骨髁的50%以上活动时,诊断得以确认。治疗包括通过修复受损结构来稳定外侧半月板后角。因此,对外侧间室进行系统的关节镜评估对于充分认识问题很重要。可通过关节镜使用各种用于半月板修复的缝合技术进行修复,效果满意且复发率低。