Vasil'eva S V, Abilev S K, Tanirbergenov T B
Genetika. 1985 Mar;21(3):364-7.
The dependence of expression of PABA antimutagenic action in bacterial cells on the character of genetic control of the mutagenic process was studied. PABA antimutagenic activity was largely connected with the negative control of SOS repair which is controlled by bacterial cell genes, but not by pKM101 plasmid genes. These results are in agreement with the idea that the systems of repair and mutagenesis specified by cell genome and plasmids are not identical.
研究了细菌细胞中对氨基苯甲酸（PABA）抗诱变作用的表达对诱变过程遗传控制特征的依赖性。PABA的抗诱变活性在很大程度上与SOS修复的负调控有关，SOS修复由细菌细胞基因而非pKM101质粒基因控制。这些结果与细胞基因组和质粒所指定的修复和诱变系统不同这一观点一致。
