T cell approach to demyelinating diseases.
作者信息
Wekerle H, Fierz W
出版信息
Springer Semin Immunopathol. 1985;8(1-2):97-110. doi: 10.1007/BF00197249.
Abstract
摘要
相似文献
1
T cell approach to demyelinating diseases.针对脱髓鞘疾病的T细胞疗法。
Springer Semin Immunopathol. 1985;8(1-2):97-110. doi: 10.1007/BF00197249.
2
Hypothesis: antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination.假设：抗原特异性T细胞使中枢神经系统内皮细胞做好准备，以招募非特异性炎症细胞来引发自身免疫性脱髓鞘。
J Neuroimmunol. 1991 Sep;33(3):237-44. doi: 10.1016/0165-5728(91)90111-j.
3
T lymphocyte lines in the study of autoimmune disease.自身免疫性疾病研究中的T淋巴细胞系
Blut. 1984 Sep;49(3):187-94. doi: 10.1007/BF00319821.
4
Autoimmune neuropathies.
Electroencephalogr Clin Neurophysiol Suppl. 1987;39:128-34.
5
B-cell antigen receptor signaling requirements for targeting antigen to the MHC class II presentation pathway.B细胞抗原受体将抗原靶向主要组织相容性复合体II类呈递途径的信号转导要求。
Curr Opin Immunol. 2004 Jun;16(3):382-7. doi: 10.1016/j.coi.2004.03.007.
6
The blood-brain barrier in virus-induced demyelination.
Adv Exp Med Biol. 1995;383:105-16. doi: 10.1007/978-1-4615-1891-4_13.
7
Anti-class II antibody treatment in autoimmune diseases.
Immunol Ser. 1993;59:279-93.
8
[A change in the expression of histocompatibility antigens could lead to autoimmunity].
Med Clin (Barc). 1984 Apr 7;82(13):594-5.
9
T cell receptors.
Lancet. 1984 Apr 7;1(8380):776.
10
Dominance of autoreactive T cell-mediated delayed-type hypersensitivity or antibody-mediated demyelination results in distinct forms of experimental autoimmune neuritis in the Lewis rat.自身反应性T细胞介导的迟发型超敏反应或抗体介导的脱髓鞘作用占主导地位，导致Lewis大鼠出现不同形式的实验性自身免疫性神经炎。
J Neuropathol Exp Neurol. 2001 Jun;60(6):637-46. doi: 10.1093/jnen/60.6.637.
引用本文的文献
1
T-cells in the cerebrospinal fluid express a similar repertoire of inflammatory chemokine receptors in the absence or presence of CNS inflammation: implications for CNS trafficking.无论中枢神经系统（CNS）有无炎症，脑脊液中的T细胞均表达相似的炎性趋化因子受体谱：对CNS细胞转运的影响
Clin Exp Immunol. 2002 Sep;129(3):510-8. doi: 10.1046/j.1365-2249.2002.01947.x.
2
Experimental allergic encephalomyelitis: the balance between encephalitogenic T lymphocytes and demyelinating antibodies determines size and structure of demyelinated lesions.实验性变应性脑脊髓炎：致脑炎性T淋巴细胞与脱髓鞘抗体之间的平衡决定了脱髓鞘病变的大小和结构。
Acta Neuropathol. 1988;75(6):566-76. doi: 10.1007/BF00686201.
3
T-cell activation by autologous human T-cell leukemia virus type I-infected T-cell clones.由自体I型人类T细胞白血病病毒感染的T细胞克隆激活T细胞
Proc Natl Acad Sci U S A. 1992 Mar 15;89(6):2110-4. doi: 10.1073/pnas.89.6.2110.
本文引用的文献
1
In vivo effect of sera from animals with chronic relapsing experimental allergic encephalomyelitis on central and peripheral myelin.慢性复发性实验性变应性脑脊髓炎动物血清对中枢和外周髓磷脂的体内效应
Acta Neuropathol. 1981;55(4):297-306. doi: 10.1007/BF00690994.
2
Chronologic neuropathology of relapsing experimental allergic encephalomyelitis in the mouse.小鼠复发性实验性变应性脑脊髓炎的时间顺序性神经病理学
Lab Invest. 1982 Feb;46(2):171-85.
3
4
Identification of T cell subsets and B lymphocytes in mouse brain experimental allergic encephalitis lesions.小鼠脑实验性变应性脑脊髓炎病变中T细胞亚群和B淋巴细胞的鉴定。
J Immunol. 1982 Oct;129(4):1649-51.
5
Vaccination against autoimmune encephalomyelitis with T-lymphocyte line cells reactive against myelin basic protein.用针对髓鞘碱性蛋白的T淋巴细胞系细胞对自身免疫性脑脊髓炎进行疫苗接种。
Nature. 1981 Jul 2;292(5818):60-1. doi: 10.1038/292060a0.
6
Complement activation by isolated myelin: activation of the classical pathway in the absence of myelin-specific antibodies.分离的髓磷脂激活补体：在没有髓磷脂特异性抗体的情况下激活经典途径。
Proc Natl Acad Sci U S A. 1982 May;79(10):3290-4. doi: 10.1073/pnas.79.10.3290.
7
A cell-surface molecule involved in organ-specific homing of lymphocytes.一种参与淋巴细胞器官特异性归巢的细胞表面分子。
Nature. 1983;304(5921):30-4. doi: 10.1038/304030a0.
8
Immunohistological analysis of T lymphocyte subsets in the central nervous system in chronic progressive multiple sclerosis.慢性进行性多发性硬化症中枢神经系统中T淋巴细胞亚群的免疫组织学分析
J Neurol Sci. 1983 Dec;62(1-3):219-32. doi: 10.1016/0022-510x(83)90201-0.
9
Interaction of T lymphocytes and macrophages with cultured vascular endothelial cells: attachment, invasion, and subsequent degradation of the subendothelial extracellular matrix.T淋巴细胞和巨噬细胞与培养的血管内皮细胞的相互作用：附着、侵袭以及随后对内皮细胞下层细胞外基质的降解。
J Cell Physiol. 1984 Feb;118(2):169-78. doi: 10.1002/jcp.1041180209.
Zotero 插件