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使用己糖激酶 2 置换肽作为治疗恶性外周神经鞘瘤的抗肿瘤方法。

The Use of Hexokinase 2-Displacing Peptides as an Anti-Neoplastic Approach for Malignant Peripheral Nerve Sheath Tumors.

机构信息

Department of Biomedical Sciences, University of Padova, 35131 Padova, Italy.

Institute of Neuroscience, National Research Council (CNR), 35131 Padova, Italy.

出版信息

Cells. 2024 Jul 8;13(13):1162. doi: 10.3390/cells13131162.

DOI:10.3390/cells13131162
PMID:38995012
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11240344/
Abstract

Malignant Peripheral Nerve Sheath Tumors (MPNSTs) are aggressive sarcomas that can arise both sporadically and in patients with the genetic syndrome Neurofibromatosis type 1 (NF1). Prognosis is dismal, as large dimensions, risk of relapse, and anatomical localization make surgery poorly effective, and no therapy is known. Hence, the identification of MPNST molecular features that could be hit in an efficient and selective way is mandatory to envision treatment options. Here, we find that MPNSTs express high levels of the glycolytic enzyme Hexokinase 2 (HK2), which is known to shield cancer cells from noxious stimuli when it localizes at MAMs (mitochondria-associated membranes), contact sites between mitochondria and endoplasmic reticulum. A HK2-targeting peptide that dislodges HK2 from MAMs rapidly induces a massive death of MPNST cells. After identifying different matrix metalloproteases (MMPs) expressed in the MPNST microenvironment, we have designed HK2-targeting peptide variants that harbor cleavage sites for these MMPs, making such peptides activatable in the proximity of cancer cells. We find that the peptide carrying the MMP2/9 cleavage site is the most effective, both in inhibiting the in vitro tumorigenicity of MPNST cells and in hampering their growth in mice. Our data indicate that detaching HK2 from MAMs could pave the way for a novel anti-MPNST therapeutic strategy, which could be flexibly adapted to the protease expression features of the tumor microenvironment.

摘要

恶性外周神经鞘瘤(MPNST)是一种侵袭性肉瘤,可在散发性和神经纤维瘤病 1 型(NF1)患者中发生。由于大尺寸、复发风险和解剖定位使手术效果不佳,且尚无已知的治疗方法,因此预后较差。因此,必须确定 MPNST 的分子特征,以便以有效和选择性的方式对其进行靶向治疗,从而为治疗方案提供思路。在这里,我们发现 MPNST 表达高水平的糖酵解酶己糖激酶 2(HK2),当它定位于线粒体相关膜(MAMs),即线粒体和内质网之间的接触点时,HK2 可保护癌细胞免受有害刺激。一种可将 HK2 从 MAMs 上置换下来的 HK2 靶向肽可迅速诱导 MPNST 细胞大量死亡。在鉴定出 MPNST 微环境中表达的不同基质金属蛋白酶(MMPs)后,我们设计了携带这些 MMP 切割位点的 HK2 靶向肽变体,使这些肽在靠近癌细胞的地方具有活性。我们发现携带 MMP2/9 切割位点的肽最有效,既能抑制 MPNST 细胞的体外致瘤性,又能阻止其在小鼠体内的生长。我们的数据表明,将 HK2 从 MAMs 上分离下来可能为一种新的抗 MPNST 治疗策略铺平道路,该策略可根据肿瘤微环境中蛋白酶的表达特征进行灵活调整。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/2a639e780570/cells-13-01162-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/bd1a9fefcfaf/cells-13-01162-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/1ad2c1669456/cells-13-01162-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/2a639e780570/cells-13-01162-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/bd1a9fefcfaf/cells-13-01162-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/1ad2c1669456/cells-13-01162-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/241d/11240344/2a639e780570/cells-13-01162-g003.jpg

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本文引用的文献

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Cancer Discov. 2023 Mar 1;13(3):654-671. doi: 10.1158/2159-8290.CD-22-0786.
2
Single-cell multiomics identifies clinically relevant mesenchymal stem-like cells and key regulators for MPNST malignancy.单细胞多组学鉴定出具有临床相关性的间充质干细胞样细胞和影响黏液样纤维肉瘤恶性程度的关键调控因子。
Sci Adv. 2022 Nov 4;8(44):eabo5442. doi: 10.1126/sciadv.abo5442. Epub 2022 Nov 2.
3
The Need for New Treatments Targeting MPNST: The Potential of Strategies Combining MEK Inhibitors with Antiangiogenic Agents.
针对 MPNST 的新型治疗方法的需求:MEK 抑制剂与抗血管生成药物联合策略的潜力。
Clin Cancer Res. 2022 Aug 2;28(15):3185-3195. doi: 10.1158/1078-0432.CCR-21-3760.
4
Malignant peripheral nerve sheath tumor: models, biology, and translation.恶性外周神经鞘瘤:模型、生物学和转化。
Oncogene. 2022 Apr;41(17):2405-2421. doi: 10.1038/s41388-022-02290-1. Epub 2022 Apr 7.
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Tumorigenesis in neurofibromatosis type 1: role of the microenvironment.神经纤维瘤病 1 型中的肿瘤发生:微环境的作用。
Oncogene. 2021 Sep;40(39):5781-5787. doi: 10.1038/s41388-021-01979-z. Epub 2021 Aug 3.
6
Hexokinase 2 in Cancer: A Prima Donna Playing Multiple Characters.己糖激酶 2 在癌症中的作用:一个扮演多个角色的主角。
Int J Mol Sci. 2021 Apr 29;22(9):4716. doi: 10.3390/ijms22094716.
7
Hexokinase 2 displacement from mitochondria-associated membranes prompts Ca -dependent death of cancer cells.己糖激酶 2 从线粒体相关膜上的移位引发依赖 Ca2+的癌细胞死亡。
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8
An Update on Patents Covering Agents That Interfere with the Cancer Glycolytic Cascade.关于干扰癌症糖酵解级联的药物的专利的最新进展。
ChemMedChem. 2018 Nov 6;13(21):2251-2265. doi: 10.1002/cmdc.201800447. Epub 2018 Oct 9.
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Trends Cell Biol. 2018 Feb;28(2):128-142. doi: 10.1016/j.tcb.2017.10.006. Epub 2017 Nov 27.
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Neurofibromatosis Type 1-Associated MPNST State of the Science: Outlining a Research Agenda for the Future.1型神经纤维瘤病相关恶性外周神经鞘膜瘤的科学现状:勾勒未来研究议程
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