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Tumor promotion in the liver.

作者信息

Schulte-Hermann R

出版信息

Arch Toxicol. 1985 Aug;57(3):147-58. doi: 10.1007/BF00290879.

DOI:10.1007/BF00290879
PMID:3904674
Abstract
摘要

相似文献

1
Tumor promotion in the liver.肝脏中的肿瘤促进作用。
Arch Toxicol. 1985 Aug;57(3):147-58. doi: 10.1007/BF00290879.
2
Chemical carcinogenesis: the liver as a model.化学致癌作用：以肝脏为模型
Pathol Immunopathol Res. 1986;5(1):1-28. doi: 10.1159/000157000.
3
Cell biology of hepatocarcinogenesis.肝癌发生的细胞生物学
Crit Rev Oncog. 1990;1(4):437-66.
4
Genetic instability occurs sooner than expected: promotion, progression and clonality during hepatocarcinogenesis in the rat.
Basic Life Sci. 1991;57:263-77. doi: 10.1007/978-1-4684-5994-4_23.
5
Occurrence of cell death (apoptosis) in preneoplastic and neoplastic liver cells. A sequential study.癌前和肿瘤性肝细胞中细胞死亡（凋亡）的发生。一项序贯研究。
Am J Pathol. 1984 Sep;116(3):441-6.
6
Pathogenesis of primary liver tumors induced by chemicals.化学物质诱导的原发性肝肿瘤的发病机制。
Recent Results Cancer Res. 1986;100:1-15. doi: 10.1007/978-3-642-82635-1_1.
7
Epigenetic mechanisms of liver tumor promotion.肝肿瘤促进的表观遗传机制。
Prog Clin Biol Res. 1990;331:131-45; discussion 147-8.
8
The effects of dose and duration of administration on the promotion index of phenobarbital in multistage hepatocarcinogenesis in the rat.给药剂量和持续时间对大鼠多阶段肝癌发生过程中苯巴比妥促进指数的影响。
APMIS Suppl. 1988;2:262-74.
9
Effect of dibutylnitrosamine and saccharin on glutamyl transpeptidase-positive foci and liver cancer.二丁基亚硝胺和糖精对谷氨酰转肽酶阳性病灶及肝癌的影响。
Environ Health Perspect. 1983 Apr;50:169-76. doi: 10.1289/ehp.8350169.
10
Cellular events during hepatocarcinogenesis in rats and the question of premalignancy.
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引用本文的文献

1
Phenobarbital does not reduce the concentration of thyroid hormone in CC57BR mice in which it does not promote liver tumor development.苯巴比妥不会降低CC57BR小鼠体内甲状腺激素的浓度，在这种小鼠中苯巴比妥不会促进肝脏肿瘤的发展。
Dokl Biol Sci. 2003 Nov-Dec;393:512-4. doi: 10.1023/b:dobs.0000010310.27212.a3.
2
Multiple end point procedure to evaluate risk from pesticides.评估农药风险的多终点程序
Environ Health Perspect. 1993 Oct;101 Suppl 3(Suppl 3):15-20. doi: 10.1289/ehp.93101s315.
3
Dose response for TCDD promotion of hepatocarcinogenesis in rats initiated with DEN: histologic, biochemical, and cell proliferation endpoints.

本文引用的文献

1
Proliferative liver lesions and sex steroids in rats.大鼠肝脏增殖性病变与性类固醇
Toxicol Pathol. 1982 Feb;10(2):132-143. doi: 10.1177/019262338201000216.
2
Response of liver foci in rats to hepatic tumor promoters.
Toxicol Pathol. 1982 Feb;10(2):63-68. doi: 10.1177/019262338201000209.
3
Biological markers of preneoplastic foci and neoplastic nodules in rodent liver.啮齿动物肝脏中癌前病灶和肿瘤结节的生物标志物。
Toxicol Pathol. 1982 Feb;10(2):19-34. doi: 10.1177/019262338201000206.
用二乙基亚硝胺引发的大鼠中，2,3,7,8-四氯二苯并对二恶英促进肝癌发生的剂量反应：组织学、生化和细胞增殖终点
Environ Health Perspect. 1993 Dec;101(7):634-42. doi: 10.1289/ehp.93101634.
4
Growth stimulation of primary rat hepatocytes by 2,3,7,8-tetrachlorodibenzo-p-dioxin.2,3,7,8-四氯二苯并对二恶英对原代大鼠肝细胞的生长刺激作用。
Cell Biol Toxicol. 1993 Jan-Mar;9(1):15-31. doi: 10.1007/BF00755137.
5
Biphasic modifying effect of indole-3-carbinol on diethylnitrosamine-induced preneoplastic glutathione S-transferase placental form-positive liver cell foci in Sprague-Dawley rats.吲哚 - 3 - 甲醇对二乙基亚硝胺诱导的斯普拉格 - 道利大鼠肝脏中癌前谷胱甘肽S - 转移酶胎盘型阳性肝细胞灶的双相修饰作用。
Jpn J Cancer Res. 1994 Jun;85(6):578-83. doi: 10.1111/j.1349-7006.1994.tb02399.x.
6
Cell proliferation and apoptosis in normal liver and preneoplastic foci.正常肝脏和癌前病灶中的细胞增殖与凋亡
Environ Health Perspect. 1993 Dec;101 Suppl 5(Suppl 5):87-90. doi: 10.1289/ehp.93101s587.
7
Selective induction of DNA synthesis in mouse preneoplastic and neoplastic hepatic lesions after exposure to phenobarbital.苯巴比妥暴露后小鼠肝前肿瘤性和肿瘤性病变中DNA合成的选择性诱导。
Environ Health Perspect. 1993 Dec;101 Suppl 5(Suppl 5):235-9. doi: 10.1289/ehp.93101s5235.
8
Association between responsiveness to phenobarbital induction of CYP2B1/2 and 3A1 in rat hepatic hyperplastic nodules and their zonal origin.大鼠肝增生结节中CYP2B1/2和3A1对苯巴比妥诱导的反应性与其区域起源之间的关联。
Environ Health Perspect. 1993 Dec;101 Suppl 5(Suppl 5):185-90. doi: 10.1289/ehp.93101s5185.
9
Cell culture assays for chemicals with tumor-promoting or tumor-inhibiting activity based on the modulation of intercellular communication.基于细胞间通讯调节的具有促肿瘤或抑肿瘤活性化学物质的细胞培养分析。
Cell Biol Toxicol. 1994 Apr;10(2):71-116. doi: 10.1007/BF00756491.
10
Assessment of the labelling index of cohorts of the pancreatic islet cell population in phenobarbitone-treated male rats using a double immunohistochemical technique for 5-bromo-2'-deoxyuridine and pancreatic hormones.使用针对5-溴-2'-脱氧尿苷和胰腺激素的双重免疫组织化学技术，评估苯巴比妥处理的雄性大鼠胰岛细胞群体队列的标记指数。
Arch Toxicol. 1994;69(1):52-8. doi: 10.1007/s002040050137.
4
Phenotypic Properties of Preneoplastic Rat Liver Lesions and Applications to Detection of Carcinogens and Tumor Promoters.
Toxicol Pathol. 1982 Feb;10(2):3-10. doi: 10.1177/019262338201000204.
5
Liver-cell tumours in rats fed hexachlorobenzene.
Cancer Lett. 1980 Dec;11(2):169-72. doi: 10.1016/0304-3835(80)90108-1.
6
Induction of hepatocellular carcinoma in rat liver by initial treatment with benzo(a)pyrene after partial hepatectomy and promotion by phenobarbital.部分肝切除术后先用苯并(a)芘处理诱导大鼠肝脏发生肝细胞癌，再用苯巴比妥促进其发展。
Toxicol Lett. 1980 Aug;6(3):167-71. doi: 10.1016/0378-4274(80)90186-1.
7
Microsomal mixed-function oxidase and activities of some related enzymes in hyperplastic nodules induced by long-term griseofulvin administration in mouse liver.
Cancer Res. 1980 Jul;40(7):2568-73.
8
Evidence for an epigenetic mode of action in organochlorine pesticide hepatocarcinogenicity: a lack of genotoxicity in rat, mouse, and hamster hepatocytes.有机氯农药致肝癌作用中表观遗传作用模式的证据：大鼠、小鼠和仓鼠肝细胞缺乏基因毒性。
J Toxicol Environ Health. 1981 Jul-Aug;8(1-2):121-30. doi: 10.1080/15287398109530056.
9
Inhibition of intercellular communication between liver cells by the liver tumor promoter 1,1,1-trichloro-2,2-bis(p-chlorophenyl)ethane.肝癌启动剂1,1,1-三氯-2,2-双（对氯苯基）乙烷对肝细胞间通讯的抑制作用
Cancer Lett. 1981 Feb;11(4):339-44. doi: 10.1016/0304-3835(81)90100-2.
10
Dose-dependent enhancing effect of phenobarbital on hepatocarcinogenesis initiated by diethylnitrosamine in the rat.苯巴比妥对大鼠二乙基亚硝胺引发的肝癌发生的剂量依赖性增强作用。
Gan. 1981 Feb;72(1):170-3.