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异位表达 Myomaker 和 Myomixer 在慢肌细胞中诱导慢肌融合和肌纤维死亡。

Ectopic expression of Myomaker and Myomixer in slow muscle cells induces slow muscle fusion and myofiber death.

机构信息

Department of Biochemistry and Molecular Biology, Institute of Marine and Environmental Technology, University of Maryland School of Medicine, Baltimore, MD 21202, United States of America.

Department of Biochemistry and Molecular Biology, Institute of Marine and Environmental Technology, University of Maryland School of Medicine, Baltimore, MD 21202, United States of America.

出版信息

J Genet Genomics. 2024 Nov;51(11):1187-1203. doi: 10.1016/j.jgg.2024.08.006. Epub 2024 Aug 30.

Abstract

Zebrafish embryos possess two major types of myofibers, the slow and fast fibers, with distinct patterns of cell fusion. The fast muscle cells can fuse, while the slow muscle cells cannot. Here, we show that myomaker is expressed in both slow and fast muscle precursors, whereas myomixer is exclusive to fast muscle cells. The loss of Prdm1a, a regulator of slow muscle differentiation, results in strong myomaker and myomixer expression and slow muscle cell fusion. This abnormal fusion is further confirmed by the direct ectopic expression of myomaker or myomixer in slow muscle cells of transgenic models. Using the transgenic models, we show that the heterologous fusion between slow and fast muscle cells can alter slow muscle cell migration and gene expression. Furthermore, the overexpression of myomaker and myomixer also disrupts membrane integrity, resulting in muscle cell death. Collectively, this study identifies that the fiber-type-specific expression of fusogenic proteins is critical for preventing inappropriate fusion between slow and fast fibers in fish embryos, highlighting the need for precise regulation of fusogenic gene expression to maintain muscle fiber integrity and specificity.

摘要

斑马鱼胚胎具有两种主要类型的肌纤维,即慢肌纤维和快肌纤维,它们具有不同的细胞融合模式。快肌细胞可以融合,而慢肌细胞则不能。在这里,我们表明肌生成素在慢肌和快肌前体中都有表达,而肌融合素则只存在于快肌细胞中。慢肌分化的调节因子 Prdm1a 的缺失导致肌生成素和肌融合素的强烈表达和慢肌细胞融合。这种异常融合进一步通过在转基因模型中的慢肌细胞中直接异位表达肌生成素或肌融合素来证实。利用转基因模型,我们表明慢肌和快肌细胞之间的异源融合可以改变慢肌细胞的迁移和基因表达。此外,肌生成素和肌融合素的过表达也会破坏细胞膜的完整性,导致肌肉细胞死亡。总之,这项研究表明,纤维类型特异性融合蛋白的表达对于防止鱼类胚胎中慢肌和快肌纤维之间的不当融合至关重要,这突出了精确调节融合基因表达以维持肌肉纤维完整性和特异性的必要性。

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本文引用的文献

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Cryo-EM structures of Myomaker reveal a molecular basis for myoblast fusion.肌母细胞融合的分子基础:Myomaker 的冷冻电镜结构
Nat Struct Mol Biol. 2023 Nov;30(11):1746-1754. doi: 10.1038/s41594-023-01110-8. Epub 2023 Sep 28.
6
Comparative hybridization protocols in zebrafish.斑马鱼的比较杂交方案。
Biotechniques. 2022 Sep;73(3):123-130. doi: 10.2144/btn-2022-0038. Epub 2022 Sep 6.

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