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真菌病感染中从病原体、免疫细胞和全身系统角度看代谢稳态

Metabolic homeostasis in fungal infections from the perspective of pathogens, immune cells, and whole-body systems.

机构信息

Department of Biochemistry and Molecular Biology and the Infection Program, Biomedicine Discovery Institute, Monash University, Clayton, Victoria, Australia.

Centre to Impact AMR, Monash University, Clayton, Victoria, Australia.

出版信息

Microbiol Mol Biol Rev. 2024 Sep 26;88(3):e0017122. doi: 10.1128/mmbr.00171-22. Epub 2024 Sep 4.

DOI:10.1128/mmbr.00171-22
PMID:39230301
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11426019/
Abstract

SUMMARYThe ability to overcome metabolic stress is a major determinant of outcomes during infections. Pathogens face nutrient and oxygen deprivation in host niches and during their encounter with immune cells. Immune cells require metabolic adaptations for producing antimicrobial compounds and mounting antifungal inflammation. Infection also triggers systemic changes in organ metabolism and energy expenditure that range from an enhanced metabolism to produce energy for a robust immune response to reduced metabolism as infection progresses, which coincides with immune and organ dysfunction. Competition for energy and nutrients between hosts and pathogens means that successful survival and recovery from an infection require a balance between elimination of the pathogen by the immune systems (resistance), and doing so with minimal damage to host tissues and organs (tolerance). Here, we discuss our current knowledge of pathogen, immune cell and systemic metabolism in fungal infections, and the impact of metabolic disorders, such as obesity and diabetes. We put forward the idea that, while our knowledge of the use of metabolic regulation for fungal proliferation and antifungal immune responses (i.e., resistance) has been growing over the years, we also need to study the metabolic mechanisms that control tolerance of fungal pathogens. A comprehensive understanding of how to balance resistance and tolerance by metabolic interventions may provide insights into therapeutic strategies that could be used adjunctly with antifungal drugs to improve patient outcomes.

摘要

摘要

在感染过程中,克服代谢应激的能力是决定结果的主要因素。病原体在宿主小生境中和与免疫细胞接触时会面临营养和氧气的剥夺。免疫细胞需要代谢适应来产生抗菌化合物和引发抗真菌炎症。感染还会引发器官代谢和能量消耗的全身性变化,从增强代谢以产生能量来支持强大的免疫反应,到随着感染的进展而减少代谢,这与免疫和器官功能障碍同时发生。宿主和病原体之间对能量和营养的竞争意味着,成功地从感染中存活和恢复需要在免疫系统消除病原体(抵抗力)和对宿主组织和器官的最小损伤(耐受性)之间取得平衡。在这里,我们讨论了我们目前对真菌感染中病原体、免疫细胞和全身代谢的了解,以及代谢紊乱(如肥胖和糖尿病)的影响。我们提出这样一种观点,即虽然我们对代谢调节在真菌增殖和抗真菌免疫反应中的作用(即抵抗力)的认识多年来一直在增长,但我们还需要研究控制真菌病原体耐受的代谢机制。通过代谢干预来平衡抵抗力和耐受性的全面理解可能为治疗策略提供新的见解,这些策略可以与抗真菌药物联合使用,以改善患者的预后。