Developing Krüppel-Like Factor 15 Agonists for Kidney Disease.
作者信息
Chow Andrew K, Guo Yiqing, Mallipattu Sandeep K
机构信息
Division of Nephrology, Department of Medicine, Stony Brook University, Stony Brook, New York.
Renal Section, Northport VA Medical Center, Northport, New York.
出版信息
J Am Soc Nephrol. 2024 Oct 1;35(10):1434-1437. doi: 10.1681/ASN.0000000000000417. Epub 2024 May 15.
Abstract
摘要
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本文引用的文献
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The transcriptional regulator KLF15 is necessary for myoblast differentiation and muscle regeneration by activating FKBP5.转录调节因子 KLF15 通过激活 FKBP5 对于成肌细胞分化和肌肉再生是必需的。
J Biol Chem. 2023 Oct;299(10):105226. doi: 10.1016/j.jbc.2023.105226. Epub 2023 Sep 4.
2
Krüppel-like factor 15 in liver diseases: Insights into metabolic reprogramming.肝脏疾病中的Krüppel样因子15：对代谢重编程的见解
Front Pharmacol. 2023 Mar 2;14:1115226. doi: 10.3389/fphar.2023.1115226. eCollection 2023.
3
Loss of proximal tubular transcription factor Krüppel-like factor 15 exacerbates kidney injury through loss of fatty acid oxidation.近端肾小管转录因子 Krüppel 样因子 15 的缺失通过脂肪酸氧化的丧失加剧了肾脏损伤。
Kidney Int. 2021 Dec;100(6):1250-1267. doi: 10.1016/j.kint.2021.08.031. Epub 2021 Oct 9.
4
Multiple roles of KLF15 in the heart: Underlying mechanisms and therapeutic implications.KLF15 在心脏中的多重作用：潜在机制和治疗意义。
J Mol Cell Cardiol. 2019 Apr;129:193-196. doi: 10.1016/j.yjmcc.2019.01.024. Epub 2019 Mar 2.
5
Podocyte-Specific Induction of Krüppel-Like Factor 15 Restores Differentiation Markers and Attenuates Kidney Injury in Proteinuric Kidney Disease.足细胞特异性诱导 Krüppel 样因子 15 恢复分化标志物并减轻蛋白尿性肾病的肾脏损伤。
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6
Loss of the podocyte glucocorticoid receptor exacerbates proteinuria after injury.足细胞糖皮质激素受体缺失加剧损伤后蛋白尿。
Sci Rep. 2017 Aug 29;7(1):9833. doi: 10.1038/s41598-017-10490-z.
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Development. 2017 Mar 1;144(5):737-754. doi: 10.1242/dev.145441.
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