• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Retraction Note: NO donor inhibits proliferation and induces apoptosis by targeting PI3K/AKT/mTOR and MEK/ERK pathways in hepatocellular carcinoma cells.

作者信息

Liu Ling, Chen Jingjing, Cao Mengyao, Wang Jiangang, Wang Shuying

机构信息

Department of Pharmacy, Medical College, Henan University of Science and Technology, 263 Kaiyuan Avenue, Luoyang, 471023, China.

出版信息

Cancer Chemother Pharmacol. 2024 Dec;94(6):819. doi: 10.1007/s00280-024-04718-9.

DOI:10.1007/s00280-024-04718-9
PMID:39412682
Abstract
摘要

相似文献

1
Retraction Note: NO donor inhibits proliferation and induces apoptosis by targeting PI3K/AKT/mTOR and MEK/ERK pathways in hepatocellular carcinoma cells.撤稿声明:NO供体通过靶向PI3K/AKT/mTOR和MEK/ERK信号通路抑制肝癌细胞增殖并诱导其凋亡
Cancer Chemother Pharmacol. 2024 Dec;94(6):819. doi: 10.1007/s00280-024-04718-9.
2
NO donor inhibits proliferation and induces apoptosis by targeting PI3K/AKT/mTOR and MEK/ERK pathways in hepatocellular carcinoma cells.无供体通过靶向肝癌细胞中的 PI3K/AKT/mTOR 和 MEK/ERK 通路抑制增殖并诱导细胞凋亡。
Cancer Chemother Pharmacol. 2019 Dec;84(6):1303-1314. doi: 10.1007/s00280-019-03965-5. Epub 2019 Sep 25.
3
Vertical Targeting of AKT and mTOR as Well as Dual Targeting of AKT and MEK Signaling Is Synergistic in Hepatocellular Carcinoma.在肝细胞癌中,AKT和mTOR的垂直靶向以及AKT和MEK信号通路的双重靶向具有协同作用。
J Cancer. 2015 Sep 16;6(12):1195-205. doi: 10.7150/jca.12452. eCollection 2015.
4
[Ponatinib inhibits the proliferation of SNU-449 human hepatocellular cancer cells and blocks MAPK and PDK1/AKT/mTOR signaling pathways].波纳替尼抑制SNU-449人肝癌细胞的增殖并阻断MAPK和PDK1/AKT/mTOR信号通路
Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2022 May;38(5):425-431.
5
[Cross-talk between PI3K/Akt and MEK/ERK pathways regulates human hepatocellular carcinoma cell cycle progression under endoplasmic reticulum stress].[PI3K/Akt与MEK/ERK信号通路间的相互作用在内质网应激下调控人肝癌细胞周期进程]
Zhonghua Gan Zang Bing Za Zhi. 2010 Dec;18(12):909-14. doi: 10.3760/cma.j.issn.1007-3418.2010.12.007.
6
Des-gamma-carboxy prothrombin antagonizes the effects of Sorafenib on human hepatocellular carcinoma through activation of the Raf/MEK/ERK and PI3K/Akt/mTOR signaling pathways.去γ-羧基凝血酶原通过激活Raf/MEK/ERK和PI3K/Akt/mTOR信号通路拮抗索拉非尼对人肝细胞癌的作用。
Oncotarget. 2016 Jun 14;7(24):36767-36782. doi: 10.18632/oncotarget.9168.
7
Synthetic lethal interaction between PI3K/Akt/mTOR and Ras/MEK/ERK pathway inhibition in rhabdomyosarcoma.横纹肌肉瘤中 PI3K/Akt/mTOR 和 Ras/MEK/ERK 通路抑制的合成致死相互作用。
Cancer Lett. 2013 Sep 1;337(2):200-9. doi: 10.1016/j.canlet.2013.05.010. Epub 2013 May 16.
8
Zeylenone, a naturally occurring cyclohexene oxide, inhibits proliferation and induces apoptosis in cervical carcinoma cells via PI3K/AKT/mTOR and MAPK/ERK pathways.泽仑诺酮,一种天然存在的环己烯氧化物,通过 PI3K/AKT/mTOR 和 MAPK/ERK 通路抑制宫颈癌细胞增殖并诱导细胞凋亡。
Sci Rep. 2017 May 10;7(1):1669. doi: 10.1038/s41598-017-01804-2.
9
CK-3, A Novel Methsulfonyl Pyridine Derivative, Suppresses Hepatocellular Carcinoma Proliferation and Invasion by Blocking the PI3K/AKT/mTOR and MAPK/ERK Pathways.新型甲磺酰基吡啶衍生物CK-3通过阻断PI3K/AKT/mTOR和MAPK/ERK信号通路抑制肝癌细胞的增殖和侵袭。
Front Oncol. 2021 Jul 28;11:717626. doi: 10.3389/fonc.2021.717626. eCollection 2021.
10
Targeting the PI3K/AKT/mTOR and RAF/MEK/ERK pathways for cancer therapy.靶向PI3K/AKT/mTOR和RAF/MEK/ERK信号通路进行癌症治疗。
Mol Biomed. 2022 Dec 21;3(1):47. doi: 10.1186/s43556-022-00110-2.

本文引用的文献

1
Protein phosphatase 2A activation mechanism contributes to JS-K induced caspase-dependent apoptosis in human hepatocellular carcinoma cells.蛋白磷酸酶 2A 的激活机制有助于 JS-K 诱导人肝癌细胞中的 caspase 依赖性细胞凋亡。
J Exp Clin Cancer Res. 2018 Jul 9;37(1):142. doi: 10.1186/s13046-018-0823-2.