Retraction: KRAS Activation in Gastric Adenocarcinoma Stimulates Epithelial-to-Mesenchymal Transition to Cancer Stem-Like Cells and Promotes Metastasis.
作者信息
Yoon Changhwan, Till Jacob, Cho Soo-Jeong, Chang Kevin K, Lin Jian-Xian, Huang Chang-Ming, Ryeom Sandra, Yoon Sam S
出版信息
Mol Cancer Res. 2024 Nov 1;22(11):1066. doi: 10.1158/1541-7786.MCR-24-0858.
Abstract
摘要
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Retraction: KRAS Activation in Gastric Adenocarcinoma Stimulates Epithelial-to-Mesenchymal Transition to Cancer Stem-Like Cells and Promotes Metastasis.撤稿声明：胃腺癌中的KRAS激活刺激上皮-间质转化为癌干细胞样细胞并促进转移。
Mol Cancer Res. 2024 Nov 1;22(11):1066. doi: 10.1158/1541-7786.MCR-24-0858.
2
RETRACTED: KRAS Activation in Gastric Adenocarcinoma Stimulates Epithelial-to-Mesenchymal Transition to Cancer Stem-Like Cells and Promotes Metastasis.撤回：胃腺癌中的 KRAS 激活刺激上皮-间充质转化为癌症干细胞样细胞并促进转移。
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KRAS activation in gastric cancer stem-like cells promotes tumor angiogenesis and metastasis.胃癌干细胞样细胞中的 KRAS 激活促进肿瘤血管生成和转移。
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ATG4A promotes tumor metastasis by inducing the epithelial-mesenchymal transition and stem-like properties in gastric cells.ATG4A通过诱导胃细胞的上皮-间质转化和干细胞样特性促进肿瘤转移。
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RETRACTED: Role of Rac1 Pathway in Epithelial-to-Mesenchymal Transition and Cancer Stem-like Cell Phenotypes in Gastric Adenocarcinoma.撤回：Rac1信号通路在胃腺癌上皮-间质转化及癌干细胞样表型中的作用
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High expression of vinculin predicts poor prognosis and distant metastasis and associates with influencing tumor-associated NK cell infiltration and epithelial-mesenchymal transition in gastric cancer.黏着斑蛋白高表达预示着不良预后和远处转移，并与影响胃癌肿瘤相关自然杀伤细胞浸润和上皮-间充质转化有关。
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Combination of epithelial-mesenchymal transition and cancer stem cell-like phenotypes has independent prognostic value in gastric cancer.上皮-间充质转化和癌症干细胞样表型的联合具有独立的胃癌预后价值。
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Epithelial-mesenchymal transition and gastric cancer stem cell.上皮-间质转化与胃癌干细胞
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Activation of PPARγ in Myeloid Cells Promotes Progression of Epithelial Lung Tumors through TGFβ1.髓系细胞中过氧化物酶体增殖物激活受体γ的激活通过 TGFβ1 促进上皮性肺肿瘤的进展。
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Retraction Note: KRAS activation in gastric cancer stem-like cells promotes tumor angiogenesis and metastasis.撤稿说明：胃癌干细胞样细胞中的KRAS激活促进肿瘤血管生成和转移。
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本文引用的文献
1
Lymphatic metastasis-related TBL1XR1 enhances stemness and metastasis in gastric cancer stem-like cells by activating ERK1/2-SOX2 signaling.淋巴转移相关 TBL1XR1 通过激活 ERK1/2-SOX2 信号增强胃癌肿瘤干细胞的干性和转移能力。
Oncogene. 2021 Feb;40(5):922-936. doi: 10.1038/s41388-020-01571-x. Epub 2020 Dec 7.
2
RETRACTED: CDX1 Expression Induced by CagA-Expressing Promotes Gastric Tumorigenesis.撤回：表达 CagA 的 CDX1 诱导促进胃肿瘤发生。
Mol Cancer Res. 2019 Nov;17(11):2169-2183. doi: 10.1158/1541-7786.MCR-19-0181. Epub 2019 Aug 15.
3
RETRACTED: KRAS Activation in Gastric Adenocarcinoma Stimulates Epithelial-to-Mesenchymal Transition to Cancer Stem-Like Cells and Promotes Metastasis.撤回：胃腺癌中的 KRAS 激活刺激上皮-间充质转化为癌症干细胞样细胞并促进转移。
Mol Cancer Res. 2019 Sep;17(9):1945-1957. doi: 10.1158/1541-7786.MCR-19-0077. Epub 2019 Jun 19.
4
RETRACTED: Role of Rac1 Pathway in Epithelial-to-Mesenchymal Transition and Cancer Stem-like Cell Phenotypes in Gastric Adenocarcinoma.撤回：Rac1信号通路在胃腺癌上皮-间质转化及癌干细胞样表型中的作用
Mol Cancer Res. 2017 Aug;15(8):1106-1116. doi: 10.1158/1541-7786.MCR-17-0053. Epub 2017 May 1.
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