Editorial: Role of mitochondrial stress response in metabolic health.
作者信息
Pereira Renata O, Keipert Susanne
机构信息
Fraternal Order of Eagles Diabetes Research Center and Division of Endocrinology and Metabolism, Roy J. and Lucille A. Carver College of Medicine, University of Iowa, Iowa City, IA, United States.
Department of Molecular Biosciences, The Wenner-Gren Institute, Stockholm University, Stockholm, Sweden.
出版信息
Front Endocrinol (Lausanne). 2024 Oct 17;15:1504718. doi: 10.3389/fendo.2024.1504718. eCollection 2024.
Abstract
摘要
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本文引用的文献
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Diabetes. 2024 Feb 1;73(2):151-161. doi: 10.2337/dbi23-0003.
2
GDF15 is required for cold-induced thermogenesis and contributes to improved systemic metabolic health following loss of OPA1 in brown adipocytes.GDF15 对于冷诱导产热是必需的，并有助于在棕色脂肪细胞中 OPA1 缺失后改善全身代谢健康。
Elife. 2023 Oct 11;12:e86452. doi: 10.7554/eLife.86452.
3
Stress-induced FGF21 and GDF15 in obesity and obesity resistance.应激诱导的成纤维细胞生长因子21和生长分化因子15在肥胖及肥胖抵抗中的作用
Trends Endocrinol Metab. 2021 Nov;32(11):904-915. doi: 10.1016/j.tem.2021.08.008. Epub 2021 Sep 13.
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OPA1 deletion in brown adipose tissue improves thermoregulation and systemic metabolism via FGF21.棕色脂肪组织中 OPA1 的缺失通过 FGF21 改善体温调节和全身代谢。
Elife. 2021 May 4;10:e66519. doi: 10.7554/eLife.66519.
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Muscle-derived GDF15 drives diurnal anorexia and systemic metabolic remodeling during mitochondrial stress.肌肉来源的 GDF15 在粒体应激时驱动昼夜厌食和全身代谢重塑。
EMBO Rep. 2020 Mar 4;21(3):e48804. doi: 10.15252/embr.201948804. Epub 2020 Feb 6.
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J Mol Endocrinol. 2018 Oct 1;61(3):R91-R105. doi: 10.1530/JME-18-0005.
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Am J Physiol Endocrinol Metab. 2014 Mar 1;306(5):E469-82. doi: 10.1152/ajpendo.00330.2013. Epub 2013 Dec 17.
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