揭开抗氧化剂的真相：线粒体应激反应解释了活性氧诱导的健康益处。 - Suppr | 超能文献

相似文献

1

Unraveling the truth about antioxidants: mitohormesis explains ROS-induced health benefits.揭开抗氧化剂的真相：线粒体应激反应解释了活性氧诱导的健康益处。

Nat Med. 2014 Jul;20(7):709-11. doi: 10.1038/nm.3624.

2

Mitohormesis in exercise training.运动训练中的线粒体激素作用。

Free Radic Biol Med. 2016 Sep;98:123-130. doi: 10.1016/j.freeradbiomed.2015.11.032. Epub 2015 Nov 30.

3

Mitohormesis, an Antiaging Paradigm.线粒体激素，一种抗衰老的范例。

Int Rev Cell Mol Biol. 2018;340:35-77. doi: 10.1016/bs.ircmb.2018.05.002. Epub 2018 Jun 21.

4

Mitohormesis; Potential implications in neurodegenerative diseases.线粒体激素；在神经退行性疾病中的潜在影响。

Mitochondrion. 2021 Jan;56:40-46. doi: 10.1016/j.mito.2020.11.011. Epub 2020 Nov 19.

5

How increased oxidative stress promotes longevity and metabolic health: The concept of mitochondrial hormesis (mitohormesis).氧化应激如何促进长寿和代谢健康：线粒体应激（mitohormesis）的概念。

Exp Gerontol. 2010 Jun;45(6):410-8. doi: 10.1016/j.exger.2010.03.014. Epub 2010 Mar 27.

6

Importance of oxidative damage on the electron transport chain for the rational use of mitochondria-targeted antioxidants.氧化损伤对电子传递链的重要性及其在线粒体靶向抗氧化剂合理应用中的意义。

Mini Rev Med Chem. 2011 Jun;11(7):625-32. doi: 10.2174/138955711795906879.

7

Sublethal mitochondrial stress with an attendant stoichiometric augmentation of reactive oxygen species may precipitate many of the beneficial alterations in cellular physiology produced by caloric restriction, intermittent fasting, exercise and dietary phytonutrients: "Mitohormesis" for health and vitality.伴有活性氧化学计量增加的亚致死性线粒体应激可能会引发热量限制、间歇性禁食、运动和膳食植物营养素所产生的许多细胞生理学有益改变：即促进健康与活力的“线粒体应激反应”。

Med Hypotheses. 2006;66(4):832-43. doi: 10.1016/j.mehy.2005.09.009. Epub 2005 Oct 18.

8

Mitochondria: Targeting mitochondrial reactive oxygen species with mitochondriotropic polyphenolic-based antioxidants.线粒体：靶向线粒体活性氧的具有线粒体靶向性的多酚类抗氧化剂。

Int J Biochem Cell Biol. 2018 Apr;97:98-103. doi: 10.1016/j.biocel.2018.02.007. Epub 2018 Feb 14.

9

Reductive stress impairs myoblasts mitochondrial function and triggers mitochondrial hormesis.还原应激会损害成肌细胞的线粒体功能并引发线粒体应激反应。

Biochim Biophys Acta. 2015 Jul;1853(7):1574-85. doi: 10.1016/j.bbamcr.2015.03.006. Epub 2015 Mar 10.

10

Strategies to target mitochondria and oxidative stress by antioxidants: key points and perspectives.抗氧化剂靶向线粒体和氧化应激的策略：要点与展望。

Pharm Res. 2011 Nov;28(11):2771-9. doi: 10.1007/s11095-011-0587-2. Epub 2011 Sep 15.

引用本文的文献

1

A Reassessment of Sarcopenia from a Redox Perspective as a Basis for Preventive and Therapeutic Interventions.从氧化还原角度对肌肉减少症进行重新评估，作为预防和治疗干预的基础。

Int J Mol Sci. 2025 Aug 12;26(16):7787. doi: 10.3390/ijms26167787.

2

Exploring the Antioxidant Roles of Cysteine and Selenocysteine in Cellular Aging and Redox Regulation.探索半胱氨酸和硒代半胱氨酸在细胞衰老和氧化还原调节中的抗氧化作用。

Biomolecules. 2025 Aug 3;15(8):1115. doi: 10.3390/biom15081115.

3

Inter-Organelle Crosstalk in Oxidative Distress: A Unified TRPM2-NOX2 Mediated Vicious Cycle Involving Ca, Zn, and ROS Amplification.

本文引用的文献

1

Metformin promotes lifespan through mitohormesis via the peroxiredoxin PRDX-2.二甲双胍通过过氧化物酶 PRDX-2 介导的 mitohormesis 促进寿命。

Proc Natl Acad Sci U S A. 2014 Jun 17;111(24):E2501-9. doi: 10.1073/pnas.1321776111. Epub 2014 Jun 2.

2

Sestrin2 inhibits uncoupling protein 1 expression through suppressing reactive oxygen species.硒蛋白2通过抑制活性氧来抑制解偶联蛋白1的表达。

Proc Natl Acad Sci U S A. 2014 May 27;111(21):7849-54. doi: 10.1073/pnas.1401787111. Epub 2014 May 13.

3

D-Glucosamine supplementation extends life span of nematodes and of ageing mice.