Johnston C E, Happel L T, Norris R, Burke S W, King A G, Roberts J M
J Bone Joint Surg Am. 1986 Apr;68(4):556-63.
The cases of two patients with delayed paraplegia after segmental spinal instrumentation with sublaminar wiring are reported. Both patients had complex spinal deformities and had transient neural deficits after the first-stage procedure of anterior release and spine fusion. They had uneventful spinal-cord monitoring during the second-stage procedure of posterior instrumentation and fusion, and function of the lower extremities was present immediately after that operation. Paraplegia then ensued, and was recognized thirty hours later in one patient and six days later in the other. Considering our reproducible and reliable experience (no false-negative results) with spinal cord monitoring in 307 operations, we propose that the delayed onset of paraplegia resulted from a progression of ischemic and edema-producing events that had not developed sufficiently intraoperatively to be reflected by the monitoring. The paraplegia became evident only when the subarachnoid space was obstructed because of progressive postoperative neural edema. The presence of sublaminar implants in narrow, kyphotic segments of the spinal canal probably exacerbated the neural irritation by dural impingement, which was seen myelographically.
报告了两例采用椎板下穿线进行节段性脊柱内固定术后出现迟发性截瘫的病例。两名患者均患有复杂的脊柱畸形,在一期前路松解和脊柱融合手术后均出现短暂性神经功能缺损。在二期后路内固定和融合手术过程中,他们的脊髓监测均无异常,术后下肢功能立即恢复。随后发生了截瘫,其中一名患者在术后30小时被发现,另一名患者在术后6天被发现。考虑到我们在307例手术中进行脊髓监测的可重复性和可靠性经验(无假阴性结果),我们认为迟发性截瘫是由缺血和产生水肿的事件进展所致,这些事件在术中未充分发展,以至于监测未能反映出来。只有当术后神经水肿逐渐加重导致蛛网膜下腔阻塞时,截瘫才变得明显。椎管狭窄、后凸节段中椎板下植入物的存在可能通过硬膜撞击加剧了神经刺激,这在脊髓造影中可见。