Passamonte P M, Dix J D
Am J Med Sci. 1985 Feb;289(2):65-7. doi: 10.1097/00000441-198502000-00005.
We postulate that the previously healthy woman reported here developed abnormal host defense mechanisms because of acute renal failure, metabolic acidosis, hyperglycemia, and glucocorticosteroid administration. Pneumonia unresponsive to antibiotics terminated in massive fatal hemoptysis that was due to mucormycosis with rupture of the pulmonary artery into the tracheobronchial tree.
我们推测,本文报道的这位既往健康的女性由于急性肾衰竭、代谢性酸中毒、高血糖及糖皮质激素的使用而出现了异常的宿主防御机制。对抗生素治疗无反应的肺炎最终导致了大量致命性咯血,这是由毛霉菌病引起的,肺动脉破裂进入气管支气管树所致。