评论：阻断B细胞活化因子可减轻右旋糖酐硫酸钠诱导的慢性结肠炎，抑制NLRP3炎性小体和核因子κB的激活。 - Suppr

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超能文献

Commentary: BAFF blockade attenuates DSS-induced chronic colitis inhibiting NLRP3 inflammasome and NF-кB activation.

作者信息

Guo Jie, Li Chen-Guang

机构信息

School of Medicine, Shenzhen University, Shenzhen, China.

Pain Department of Shenzhen Nanshan People's Hospital, Shenzhen, China.

出版信息

Front Immunol. 2024 Dec 24;15:1528731. doi: 10.3389/fimmu.2024.1528731. eCollection 2024.

DOI:10.3389/fimmu.2024.1528731

PMID:39776908

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11703703/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6c33/11703703/611c633f0b19/fimmu-15-1528731-g001.jpg

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Ecotoxicol Environ Saf. 2023 Jun 15;258:114975. doi: 10.1016/j.ecoenv.2023.114975. Epub 2023 May 4.

Lipid peroxidation increases membrane tension, Piezo1 gating, and cation permeability to execute ferroptosis.脂质过氧化作用会增加膜张力，开启 Piezo1 通道，并增加阳离子通透性，从而执行铁死亡。

Curr Biol. 2023 Apr 10;33(7):1282-1294.e5. doi: 10.1016/j.cub.2023.02.060. Epub 2023 Mar 9.

Myeloid-cell-specific role of Gasdermin D in promoting lung cancer progression in mice.Gasdermin D在小鼠肺癌进展中促进作用的髓系细胞特异性作用

iScience. 2023 Jan 31;26(2):106076. doi: 10.1016/j.isci.2023.106076. eCollection 2023 Feb 17.

Front Immunol. 2022 Mar 7;13:783254. doi: 10.3389/fimmu.2022.783254. eCollection 2022.

NLRC4 inflammasome-dependent cell death occurs by a complementary series of three death pathways and determines lethality in mice.NLRC4炎性小体依赖性细胞死亡通过一系列互补的三种死亡途径发生，并决定小鼠的致死率。

Sci Adv. 2021 Oct 22;7(43):eabi9471. doi: 10.1126/sciadv.abi9471.

Propofol directly induces caspase-1-dependent macrophage pyroptosis through the NLRP3-ASC inflammasome.异丙酚通过 NLRP3-ASC 炎性小体直接诱导 caspase-1 依赖性巨噬细胞焦亡。

Cell Death Dis. 2019 Jul 17;10(8):542. doi: 10.1038/s41419-019-1761-4.

PtdIns4P on dispersed trans-Golgi network mediates NLRP3 inflammasome activation.分散的高尔基网络上的 PtdIns4P 介导 NLRP3 炎性小体的激活。

Nature. 2018 Dec;564(7734):71-76. doi: 10.1038/s41586-018-0761-3. Epub 2018 Nov 28.

Pyroptosis is driven by non-selective gasdermin-D pore and its morphology is different from MLKL channel-mediated necroptosis.细胞焦亡由非选择性的gasdermin-D孔道驱动，其形态不同于MLKL通道介导的坏死性凋亡。

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