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[出生后个体发育过程中奎尼丁在脑内的分布与心肌对乌头碱和氯化钙致心律失常易感性之间的相互关系]

[Interrelations between quinidine distribution in the brain and myocardial arrhythmogenic readiness to aconitine and calcium chloride in postnatal ontogeny].

作者信息

Kuz'min A F, Saliaev V N

出版信息

Farmakol Toksikol. 1985 Mar-Apr;48(2):59-63.

PMID:3996573
Abstract

It has been shown in experiments on rats of different age that antiarrhythmic activity of quinidine during aconitine and calcium chloride arrhythmias is mediated not only via the cardial mechanisms but also via the central nervous system, with the importance of these mechanisms in postnatal ontogenesis being inconclusive.

摘要

在对不同年龄大鼠的实验中已表明，奎尼丁在乌头碱和氯化钙所致心律失常期间的抗心律失常活性不仅通过心脏机制介导，还通过中枢神经系统介导，而这些机制在出生后个体发育中的重要性尚无定论。

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Farmakol Toksikol. 1985 Mar-Apr;48(2):59-63.
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