Bergelson L D, Kulikov V I, Muzia G I
FEBS Lett. 1985 Oct 14;190(2):305-6. doi: 10.1016/0014-5793(85)81306-5.
Prostaglandin E1 (PGE1) and platelet activation factor (PAF) were found respectively to activate or to inhibit cholesterol esterification in whole plasma but not in lipoprotein-deficient plasma. It is suggested that these effects are mediated by the interaction of PGE1 and PAF with high-density lipoproteins [(1984) FEBS Lett. 173, 291-294]. Possible physiological implications of these findings are discussed.
前列腺素E1（PGE1）和血小板活化因子（PAF）分别被发现可激活或抑制全血中的胆固醇酯化，但对脂蛋白缺乏的血浆则无此作用。提示这些效应是由PGE1和PAF与高密度脂蛋白相互作用介导的[（1984年）欧洲生物化学学会联合会快报173，291 - 294]。文中讨论了这些发现可能的生理意义。
