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[ω-3脂肪酸在杜氏肌营养不良症中的益处]

[Benefits of ω-3 fatty acids in Duchenne muscular dystrophy].

作者信息

Galeazzi-Aguilar José Emilio, Rodríguez-Cruz Maricela

机构信息

Universidad Nacional Autónoma de México, Facultad de Medicina, Posgrado en Ciencias Biomédicas. Ciudad de México, México.

Instituto Mexicano del Seguro Social, Centro Médico Nacional Siglo XXI, Hospital de Pediatría "Dr. Silvester Frenk Freund", Unidad de Investigación Médica en Nutrición. Ciudad de México, México.

出版信息

Rev Med Inst Mex Seguro Soc. 2025 Jan 2;63(Suppl 1):e6316. doi: 10.5281/zenodo.14199927.

DOI:10.5281/zenodo.14199927
PMID:40489651
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12162134/
Abstract

Duchenne Muscular Dystrophy (DMD), caused by mutations in the dystrophin gene, is the most common congenital muscular dystrophy and is characterized by a chronic inflammatory response mediated by immune cells and cytokines. Obesity, a common feature of DMD, exacerbates this inflammation. Although corticosteroids are the conventional treatment, their adverse effects are substantial. The use of omega-3 polyunsaturated fatty acids (PUFAω-3) as adjuvant therapy is under investigation. A comprehensive review of studies from 2011 to May 2024 reveals that supplementation with PUFAω-3 has beneficial effects in mdx mice, a model of DMD. These include reduction of the inflammatory process in both muscle and circulation, in addition to decreased myonecrosis, resulting in improved muscle strength and endurance. Supplementation also had a positive impact on cardiac tissue, reducing fibrosis and inflammation present in this tissue. These results were corroborated in patients with DMD. A significant decrease in inflammatory markers such as NF-kB, IL-1β and IL-6 was observed with PUFAω-3, and an increase in the anti-inflammatory cytokine IL-10, suggesting a potential anti-inflammatory effect. These findings support the use of PUFAω-3 as adjuvant therapy in DMD, although further research is needed to fully understand its mechanisms and clinical benefits.

摘要

杜兴氏肌营养不良症(DMD)由肌营养不良蛋白基因突变引起,是最常见的先天性肌营养不良症,其特征是由免疫细胞和细胞因子介导的慢性炎症反应。肥胖是DMD的一个常见特征,会加剧这种炎症。虽然皮质类固醇是传统治疗方法,但其副作用很大。ω-3多不饱和脂肪酸(PUFAω-3)作为辅助治疗的应用正在研究中。对2011年至2024年5月的研究进行的全面综述表明,补充PUFAω-3对mdx小鼠(一种DMD模型)有有益作用。这些作用包括减少肌肉和循环中的炎症过程,此外还减少了肌坏死,从而提高了肌肉力量和耐力。补充PUFAω-3对心脏组织也有积极影响,减少了该组织中的纤维化和炎症。这些结果在DMD患者中得到了证实。使用PUFAω-3后,观察到炎症标志物如NF-κB、IL-1β和IL-6显著减少,抗炎细胞因子IL-10增加,表明其具有潜在抗炎作用。这些发现支持将PUFAω-3用作DMD的辅助治疗,尽管需要进一步研究以充分了解其机制和临床益处。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6c12/12162134/382d83071bdb/04435117-63-Suppl1-e6316-f001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6c12/12162134/382d83071bdb/04435117-63-Suppl1-e6316-f001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6c12/12162134/382d83071bdb/04435117-63-Suppl1-e6316-f001.jpg

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本文引用的文献

1
The muscle regeneration marker FOXP3 is associated with muscle injury in Duchenne muscular dystrophy.肌肉再生标志物 FOXP3 与杜氏肌营养不良症中的肌肉损伤有关。
Brain Dev. 2024 May;46(5):199-206. doi: 10.1016/j.braindev.2024.02.001. Epub 2024 Feb 22.
2
Topical corticosteroids inhibit allergic skin inflammation but are ineffective in impeding the formation and expansion of resident memory T cells.局部皮质类固醇抑制过敏皮肤炎症,但在阻碍常驻记忆 T 细胞的形成和扩增方面无效。
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杜氏肌营养不良症:发病机制和有前途的治疗方法。
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Identification of immune-related features involved in Duchenne muscular dystrophy: A bidirectional transcriptome and proteome-driven analysis.鉴定杜氏肌营养不良症中的免疫相关特征:一个双向转录组和蛋白质组驱动的分析。
Front Immunol. 2022 Nov 22;13:1017423. doi: 10.3389/fimmu.2022.1017423. eCollection 2022.
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Role of Matrix Metalloproteinases in Musculoskeletal Diseases.基质金属蛋白酶在肌肉骨骼疾病中的作用。
Biomedicines. 2022 Oct 4;10(10):2477. doi: 10.3390/biomedicines10102477.
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Efficacy of the omega-3 fatty acids supplementation on inflammatory biomarkers: An umbrella meta-analysis.ω-3 脂肪酸补充剂对炎症生物标志物的疗效:伞式荟萃分析。
Int Immunopharmacol. 2022 Oct;111:109104. doi: 10.1016/j.intimp.2022.109104. Epub 2022 Jul 30.
7
Identification of Auxiliary Biomarkers and Description of the Immune Microenvironmental Characteristics in Duchenne Muscular Dystrophy by Bioinformatical Analysis and Experiment.通过生物信息学分析和实验鉴定杜兴氏肌肉营养不良症的辅助生物标志物并描述免疫微环境特征
Front Neurosci. 2022 Jun 3;16:891670. doi: 10.3389/fnins.2022.891670. eCollection 2022.
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Role of EPA in Inflammation: Mechanisms, Effects, and Clinical Relevance.EPA 在炎症中的作用:机制、影响和临床相关性。
Biomolecules. 2022 Feb 1;12(2):242. doi: 10.3390/biom12020242.
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Global prevalence of Duchenne and Becker muscular dystrophy: a systematic review and meta-analysis.全球杜氏肌营养不良症和贝克肌营养不良症的患病率:系统评价和荟萃分析。
J Orthop Surg Res. 2022 Feb 15;17(1):96. doi: 10.1186/s13018-022-02996-8.
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