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本文引用的文献

1
Intercepting Modes of Cellular Communication.细胞通讯的拦截模式
Cell Mol Gastroenterol Hepatol. 2025 May 8;19(8):101531. doi: 10.1016/j.jcmgh.2025.101531.
2
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Cell Mol Gastroenterol Hepatol. 2025 May 8;19(8):101530. doi: 10.1016/j.jcmgh.2025.101530.
3
Intestinal Stearoyl-CoA Desaturase-1 Regulates Energy Balance via Alterations in Bile Acid Homeostasis.肠酰基辅酶 A 去饱和酶 1 通过改变胆汁酸稳态调节能量平衡。
Cell Mol Gastroenterol Hepatol. 2024;18(6):101403. doi: 10.1016/j.jcmgh.2024.101403. Epub 2024 Sep 14.
4
Microbial-derived Urolithin A Targets GLS1 to Inhibit Glutaminolysis and Attenuate Cirrhotic Portal Hypertension.微生物源性尿石素 A 靶向 GLS1 抑制谷氨酰胺分解代谢并减轻肝硬化门脉高压。
Cell Mol Gastroenterol Hepatol. 2024;18(4):101379. doi: 10.1016/j.jcmgh.2024.101379. Epub 2024 Jul 20.
5
Hepatocellular RECK as a Critical Regulator of Metabolic Dysfunction-associated Steatohepatitis Development.肝细胞 REck 作为代谢功能障碍相关脂肪性肝炎发展的关键调节因子。
Cell Mol Gastroenterol Hepatol. 2024;18(3):101365. doi: 10.1016/j.jcmgh.2024.101365. Epub 2024 May 24.
6
Endothelial Slc35a1 Deficiency Causes Loss of LSEC Identity and Exacerbates Neonatal Lipid Deposition in the Liver in Mice.内皮细胞 Slc35a1 缺乏导致小鼠肝脏中 LSEC 特征丧失，并加剧新生儿期脂质沉积。
Cell Mol Gastroenterol Hepatol. 2024;17(6):1039-1061. doi: 10.1016/j.jcmgh.2024.03.002. Epub 2024 Mar 11.
7
Fibroblast Growth Factor 19 Alters Bile Acids to Induce Dysbiosis in Mice With Alcohol-Induced Liver Disease.成纤维细胞生长因子 19 通过改变胆汁酸在酒精性肝病小鼠中诱导菌群失调。
Cell Mol Gastroenterol Hepatol. 2024;18(1):71-87. doi: 10.1016/j.jcmgh.2024.02.015. Epub 2024 Feb 28.
8
Advances in Brain-Gut-Microbiome Interactions: A Comprehensive Update on Signaling Mechanisms, Disorders, and Therapeutic Implications.脑-肠-微生物组相互作用的研究进展：信号机制、疾病及治疗意义的综合更新。
Cell Mol Gastroenterol Hepatol. 2024;18(1):1-13. doi: 10.1016/j.jcmgh.2024.01.024. Epub 2024 Feb 8.
9
A Structure-function Analysis of Hepatocyte Arginase 2 Reveals Mitochondrial Ureahydrolysis as a Determinant of Glucose Oxidation.肝细胞精氨酸酶 2 的结构-功能分析揭示了线粒体脲水解作为葡萄糖氧化的决定因素。
Cell Mol Gastroenterol Hepatol. 2024;17(5):801-820. doi: 10.1016/j.jcmgh.2024.01.016. Epub 2024 Jan 25.
10
Oral Supplementation of Phosphatidylcholine Attenuates the Onset of a Diet-Induced Metabolic Dysfunction-Associated Steatohepatitis in Female C57BL/6J Mice.口服补充磷脂酰胆碱可减轻饮食诱导的代谢功能障碍相关脂肪性肝炎在雌性 C57BL/6J 小鼠中的发生。
Cell Mol Gastroenterol Hepatol. 2024;17(5):785-800. doi: 10.1016/j.jcmgh.2024.01.009. Epub 2024 Jan 21.

Transcellular, Interorgan, and Host-microbial Communication in Metabolic Homeostasis: Hearing for the First Time What We Once Thought to be Unspoken.

作者信息

Hung Lin Yung, DeBosch Brian

机构信息

Division of Pediatric Gastroenterology, Hepatology, and Nutrition, Columbia University Medical Center, New York, New York; Department of Molecular Pathobiology, New York University College of Dentistry, New York, New York.

Department of Pediatrics, Indiana University School of Medicine, Indianapolis, Indiana; Department of Anatomy, Cell Biology, and Physiology, Indiana University School of Medicine, Indianapolis, Indiana.

出版信息

Cell Mol Gastroenterol Hepatol. 2025 Jun 9;19(9):101545. doi: 10.1016/j.jcmgh.2025.101545.

DOI:10.1016/j.jcmgh.2025.101545
PMID:40532271
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12213667/
Abstract
摘要