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Molecular and biological approach to ischemia/reperfusion in diabetes conditions.

作者信息

Kilic Melis, Halici Zekai

机构信息

Department of Pharmacology, Faculty of Medicine, Atatürk University, Erzurum, 25240, Turkey.

出版信息

Mol Biol Rep. 2025 Jun 21;52(1):620. doi: 10.1007/s11033-025-10686-x.

DOI:10.1007/s11033-025-10686-x
PMID:40544201
Abstract
摘要

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本文引用的文献

1
Dexmedetomidine alleviates blood-brain barrier disruption in rats after cerebral ischemia-reperfusion by suppressing JNK and p38 MAPK signaling.右美托咪定通过抑制JNK和p38 MAPK信号通路减轻大鼠脑缺血再灌注后的血脑屏障破坏。
Korean J Physiol Pharmacol. 2024 May 1;28(3):239-252. doi: 10.4196/kjpp.2024.28.3.239.
2
Ferroptosis Is Involved in Diabetes Myocardial Ischemia/Reperfusion Injury Through Endoplasmic Reticulum Stress.铁死亡通过内质网应激参与糖尿病心肌缺血/再灌注损伤。
DNA Cell Biol. 2020 Feb;39(2):210-225. doi: 10.1089/dna.2019.5097. Epub 2019 Dec 6.
3
Sustained ER stress promotes hyperglycemia by increasing glucagon action through the deubiquitinating enzyme USP14.
持续的内质网应激通过去泛素化酶 USP14 增加胰高血糖素的作用来促进高血糖。
Proc Natl Acad Sci U S A. 2019 Oct 22;116(43):21732-21738. doi: 10.1073/pnas.1907288116. Epub 2019 Oct 8.
4
Co-morbidities and co-medications as confounders of cardioprotection-Does it matter in the clinical setting?合并症和联合用药作为心脏保护的混杂因素——在临床环境中这重要吗？
Br J Pharmacol. 2020 Dec;177(23):5252-5269. doi: 10.1111/bph.14839. Epub 2020 Jan 3.
5
Molecular basis of cardioprotection: signal transduction in ischemic pre-, post-, and remote conditioning.心肌保护的分子基础：缺血预处理、后处理和远程处理中的信号转导。
Circ Res. 2015 Feb 13;116(4):674-99. doi: 10.1161/CIRCRESAHA.116.305348.