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Myocardial reperfusion: a double-edged sword?

作者信息

Braunwald E, Kloner R A

出版信息

J Clin Invest. 1985 Nov;76(5):1713-9. doi: 10.1172/JCI112160.

DOI:10.1172/JCI112160
PMID:4056048
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC424191/
Abstract
摘要

相似文献

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Myocardial reperfusion: a double-edged sword?心肌再灌注:一把双刃剑?
J Clin Invest. 1985 Nov;76(5):1713-9. doi: 10.1172/JCI112160.
2
Pathophysiology of myocardial reperfusion.
Cardiol Clin. 1987 Feb;5(1):31-48.
3
[Reperfusion and postconditioning in acute ST segment elevation myocardial infarction. A new paradigm for the treatment of acute myocardial infarction. From bench to bedside?].[急性ST段抬高型心肌梗死的再灌注与后适应。急性心肌梗死治疗的新范例。从实验台到病床边?]
Arch Cardiol Mex. 2006 Oct-Dec;76 Suppl 4:S76-101.
4
[Myocardial damage caused by ischemia and reperfusion: role of oxygen toxicity].
Cardiologia. 1988 Jan;33(1):73-8.
5
Detection and quantification of myocardial reperfusion hemorrhage using T2*-weighted CMR.利用 T2*-加权 CMR 检测和定量心肌再灌注出血。
JACC Cardiovasc Imaging. 2011 Dec;4(12):1274-83. doi: 10.1016/j.jcmg.2011.08.016.
6
[Reperfusion during acute myocardial infarction: questions that remain obscure].[急性心肌梗死期间的再灌注:仍不明确的问题]
Arq Bras Cardiol. 1985 Jul;45(1):61-7.
7
[Reperfusion in myocardial infarction: a two-edged sword].
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8
Studies of experimental coronary artery reperfusion. Effects on infarct size, myocardial function, biochemistry, ultrastructure and microvascular damage.实验性冠状动脉再灌注研究。对梗死面积、心肌功能、生物化学、超微结构及微血管损伤的影响。
Circulation. 1983 Aug;68(2 Pt 2):I8-15.
9
Impact of perfusion restoration at epicardial and tissue levels on markers of myocardial necrosis and clinical outcome of patients with acute myocardial infarction.探讨心肌梗死患者心外膜和组织水平灌注恢复对心肌坏死标志物和临床转归的影响。
EuroIntervention. 2011 May;7(1):128-35. doi: 10.4244/EIJV7I1A21.
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[Myocardial reperfusion--a double-edged sword?].[心肌再灌注——一把双刃剑?]
Duodecim. 1989;105(9):822-30.

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本文引用的文献

1
Myocardial necrosis induced by temporary occlusion of a coronary artery in the dog.犬冠状动脉临时闭塞所致的心肌坏死
Arch Pathol. 1960 Jul;70:68-78.
2
EXPERIMENTAL ACUTE MYOCARDIAL INFARCTION; HISTOLOGIC AND HISTOCHEMICAL STUDIES OF EARLY MYOCARDIAL INFARCTS INDUCED BY TEMPORARY OR PERMANENT OCCLUSION OF A CORONARY ARTERY.实验性急性心肌梗死;冠状动脉暂时或永久性闭塞所致早期心肌梗死的组织学和组织化学研究
Lab Invest. 1964 Dec;13:1491-503.
3
LOW INCIDENCE OF CORONARY THROMBOSIS IN MYOCARDIAL INFARCTION. A RESTUDY BY SERIAL BLOCK TECHNIQUE.
Arch Pathol. 1964 Oct;78:432-45.
4
Ultrastructural evidence of microvascular damage and myocardial cell injury after coronary artery occlusion: which comes first?冠状动脉闭塞后微血管损伤和心肌细胞损伤的超微结构证据:哪个先出现?
Circulation. 1980 Nov;62(5):945-52. doi: 10.1161/01.cir.62.5.945.
5
Prevalence of total coronary occlusion during the early hours of transmural myocardial infarction.透壁性心肌梗死早期数小时内完全冠状动脉闭塞的发生率。
N Engl J Med. 1980 Oct 16;303(16):897-902. doi: 10.1056/NEJM198010163031601.
6
Effects of propranolol on myocardial infarct size with and without coronary artery reperfusion in the dog.普萘洛尔对犬冠状动脉再灌注及未再灌注时心肌梗死面积的影响。
Cardiovasc Res. 1981 Jul;15(7):411-20. doi: 10.1093/cvr/15.7.411.
7
Prolonged depletion of ATP and of the adenine nucleotide pool due to delayed resynthesis of adenine nucleotides following reversible myocardial ischemic injury in dogs.在犬可逆性心肌缺血损伤后,由于腺嘌呤核苷酸再合成延迟,导致ATP和腺嘌呤核苷酸池长期耗竭。
J Mol Cell Cardiol. 1981 Feb;13(2):229-39. doi: 10.1016/0022-2828(81)90219-4.
8
Tissue osmolality, cell swelling, and reperfusion in acute regional myocardial ischemia in the isolated porcine heart.离体猪心脏急性局部心肌缺血时的组织渗透压、细胞肿胀和再灌注
Circ Res. 1981 Aug;49(2):364-81. doi: 10.1161/01.res.49.2.364.
9
Beneficial actions of a free radical scavenger in traumatic shock and myocardial ischemia.自由基清除剂在创伤性休克和心肌缺血中的有益作用。
Circ Shock. 1981;8(3):273-82.
10
The role of lipid peroxidation in pathogenesis of ischemic damage and the antioxidant protection of the heart.脂质过氧化在缺血性损伤发病机制中的作用及心脏的抗氧化保护。
Basic Res Cardiol. 1982 Sep-Oct;77(5):465-85. doi: 10.1007/BF01907940.