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A mouse model of hypertension induced by sucrose.

作者信息

Nakayama Yuki, Utsunomiya Hirotoshi, Eguchi Kunie, Elliott Katherine J, Hashimoto Tomoki, Osei-Owusu Patrick, Eguchi Satoru

机构信息

Cardiovascular Research Center, Lewis Katz School of Medicine at Temple University, Philadelphia, PA, USA.

Department of Surgery and the Heart Institute, Morsani College of Medicine, University of South Florida, Tampa, FL, USA.

出版信息

Hypertens Res. 2025 Jul 2. doi: 10.1038/s41440-025-02278-w.

DOI:10.1038/s41440-025-02278-w
PMID:40603590
Abstract
摘要

相似文献

1
A mouse model of hypertension induced by sucrose.蔗糖诱导的高血压小鼠模型
Hypertens Res. 2025 Jul 2. doi: 10.1038/s41440-025-02278-w.
2
Cardiovascular Effects of Sucrose in Experimental Animals and Humans.
Hypertension. 2025 Aug 28. doi: 10.1161/HYPERTENSIONAHA.125.25720.
3
Effects of L-Arabinose on Glycemic Responses After the Consumption of Sucrose-Rich Foods in Individuals with Impaired Fasting Glucose: A Randomized Controlled Cross-Over Trial.
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4
The Correlation Between Angiotensin II Levels and Homeostatic Model Assessment of Insulin Resistance in Normotensive Young Adults with a Family History of Essential Hypertension.有原发性高血压家族史的血压正常年轻成年人中血管紧张素II水平与胰岛素抵抗稳态模型评估之间的相关性
Acta Med Indones. 2025 Apr;57(2):222-227.
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Chronic fructose substitution for glucose or sucrose in food or beverages has little effect on fasting blood glucose, insulin, or triglycerides: a systematic review and meta-analysis.慢性果糖替代食物或饮料中的葡萄糖或蔗糖对空腹血糖、胰岛素或甘油三酯几乎没有影响：系统评价和荟萃分析。
Am J Clin Nutr. 2017 Aug;106(2):519-529. doi: 10.3945/ajcn.116.145169. Epub 2017 Jun 7.
6
Fructose replacement of glucose or sucrose in food or beverages lowers postprandial glucose and insulin without raising triglycerides: a systematic review and meta-analysis.在食物或饮料中用果糖替代葡萄糖或蔗糖可以降低餐后血糖和胰岛素，而不会使甘油三酯升高：系统评价和荟萃分析。
Am J Clin Nutr. 2017 Aug;106(2):506-518. doi: 10.3945/ajcn.116.145151. Epub 2017 Jun 7.
7
Nintedanib inhibits neovascularization and subretinal fibrosis in a laser-induced choroidal neovascularization mouse model.在激光诱导的脉络膜新生血管小鼠模型中，尼达尼布可抑制新生血管形成和视网膜下纤维化。
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Astaxanthin improves lipotoxicity, lipid peroxidation and oxidative stress in kidney of sucrose-rich diet-fed rats.虾青素可改善高蔗糖饮食喂养大鼠肾脏中的脂毒性、脂质过氧化和氧化应激。
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Metabolic imprinting due to small litter size mitigates insulin resistance through the interscapular brown adipose tissue activation in a high-sucrose diet model.在高糖饮食模型中，由于窝仔数少导致的代谢印记通过肩胛间棕色脂肪组织激活减轻胰岛素抵抗。
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Suppressing wheat sucrose phosphate synthase 1-B protects wheat against stripe rust.抑制小麦蔗糖磷酸合成酶1-B可保护小麦免受条锈病侵害。
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本文引用的文献

1
Intake of sucrose-sweetened beverages and risk of developing pharmacologically treated hypertension in women: cohort study.女性摄入蔗糖甜味饮料与药物治疗的高血压发病风险：队列研究
BMJ Nutr Prev Health. 2022 Nov 9;5(2):277-285. doi: 10.1136/bmjnph-2022-000426. eCollection 2022 Dec.
2
Sweeteners modulate bioactivity of endothelial progenitor cells but not induce detrimental effects both on inflammation and behavioural changes.甜味剂调节内皮祖细胞的生物活性，但不会对炎症和行为变化产生不利影响。
Int J Food Sci Nutr. 2019 Sep;70(6):725-737. doi: 10.1080/09637486.2018.1563052. Epub 2019 Feb 18.
3
Angiotensin II Signal Transduction: An Update on Mechanisms of Physiology and Pathophysiology.
血管紧张素 II 信号转导：对生理和病理生理学机制的最新研究。
Physiol Rev. 2018 Jul 1;98(3):1627-1738. doi: 10.1152/physrev.00038.2017.
4
Perspective: Cardiovascular Responses to Sugar-Sweetened Beverages in Humans: A Narrative Review with Potential Hemodynamic Mechanisms.观点：人类对含糖饮料的心血管反应：一篇具有潜在血流动力学机制的叙述性综述。
Adv Nutr. 2018 Mar 1;9(2):70-77. doi: 10.1093/advances/nmx023.
5
Intake of sucrose-sweetened water induces insulin resistance and exacerbates memory deficits and amyloidosis in a transgenic mouse model of Alzheimer disease.在阿尔茨海默病转基因小鼠模型中，摄入蔗糖甜水会诱发胰岛素抵抗，并加剧记忆缺陷和淀粉样变性。
J Biol Chem. 2007 Dec 14;282(50):36275-82. doi: 10.1074/jbc.M703561200. Epub 2007 Oct 17.
6
Nocturnal hypertension in mice consuming a high fructose diet.食用高果糖饮食的小鼠的夜间高血压。
Auton Neurosci. 2006 Dec 30;130(1-2):41-50. doi: 10.1016/j.autneu.2006.05.006. Epub 2006 Jul 13.
7
Chronic sucrose ingestion induces mild hypertension and tachycardia in rats.
Hypertension. 1983 Mar-Apr;5(2):218-25. doi: 10.1161/01.hyp.5.2.218.