Abraham D
J Parasitol. 1985 Dec;71(6):742-4.
Dipetalonema viteae was studied in the jird, Meriones unguiculatus, to determine the mechanism controlling the level of peripheral microfilaremia. Jirds killed 40 days after infection served as donors of female worms of known age and reproductive status. These worms were transplanted into uninfected jirds and the resultant microfilaremias were monitored. After approximately 100 days, the recipient jirds were killed and 58% of the transplanted worms were recovered alive but depleted of sperm and microfilariae, regardless of the total number implanted in a given host. A direct linear relationship between microfilaremia and the number of recovered adult worms was found. Based on the uniform absence of sperm and microfilariae in the recovered worms it was concluded that female worms, under the conditions of the present study, do not control the peripheral microfilaremia in multi-worm infections through a reduced parturition rate.
在沙鼠（Meriones unguiculatus）体内研究了魏氏双瓣线虫（Dipetalonema viteae），以确定控制外周血微丝蚴血症水平的机制。感染后40天处死的沙鼠作为已知年龄和生殖状态的雌虫供体。将这些雌虫移植到未感染的沙鼠体内，并监测由此产生的微丝蚴血症。大约100天后，处死受体沙鼠，无论植入给定宿主的雌虫总数如何，58%的移植雌虫存活但精子和微丝蚴缺失。发现微丝蚴血症与回收的成虫数量之间存在直接线性关系。基于回收的雌虫中均不存在精子和微丝蚴，得出结论：在本研究条件下，雌虫在多虫感染中不会通过降低产仔率来控制外周血微丝蚴血症。
