Kuwano Akito, Yoshikai Masaru, Ohtsubo Satoshi, Koga Kiyokazu, Yoshida Nozomi
Department of Cardiovascular Surgery, Shin-Koga Hospital, Kurume City, Fukuoka, Japan.
J Cardiol Cases. 2025 Jun 21;32(3):123-125. doi: 10.1016/j.jccase.2025.06.001. eCollection 2025 Sep.
We report a case of aortic coarctation complicated by type B aortic dissection in an elderly patient, whose left ventricular (LV) systolic dysfunction improved after surgical repair. A 71-year-old male presented with back pain, and contrast-enhanced computed tomography (CT) revealed aortic coarctation and aortic dissection. The aortic dissection was uncomplicated type B, so the patient was managed conservatively. Transthoracic echocardiography (TTE) on admission showed eccentric hypertrophy in the Lang classification. LV wall motion was diffusely impaired, with an LV ejection fraction (EF) of 27 %. Parachute-like morphology of mitral valve and mild mitral stenosis, with an elevation of an E/e' ratio of 32.6, were also recognized. Three months later, CT revealed rapid expansion of the descending aorta, so the patient underwent resection of the coarctated segment and a graft replacement of the descending aorta. TTE three months postoperatively revealed normalization of LV systolic function, with an LVEF of 58 %, and of LV dilatation, and regression of hypertrophy, along with decrease in an E/e' ratio of 19.5. Preoperative LV systolic dysfunction seems to be attributed to afterload mismatch. Two years after the surgery, the patient remains in good health without experiencing heart failure or any aortic-related events.
The prognosis of adult aortic coarctation is poor, with the impact on cardiac function due to chronic increased afterload. We present a case of aortic coarctation in an elderly patient, whose left ventricular systolic dysfunction improved after surgical repair. We considered that surgical repair of aortic coarctation resolved eccentric hypertrophy and corrected the afterload mismatch that existed preoperatively, leading to improvement in left ventricular systolic function.
我们报告一例老年患者主动脉缩窄合并B型主动脉夹层,其左心室(LV)收缩功能障碍在手术修复后得到改善。一名71岁男性因背痛就诊,增强计算机断层扫描(CT)显示主动脉缩窄和主动脉夹层。主动脉夹层为非复杂性B型,因此对该患者进行了保守治疗。入院时经胸超声心动图(TTE)显示Lang分级为偏心性肥厚。左心室壁运动弥漫性受损,左心室射血分数(EF)为27%。还发现二尖瓣呈降落伞样形态及轻度二尖瓣狭窄,E/e'比值升高至32.6。三个月后,CT显示降主动脉迅速扩张,因此患者接受了缩窄段切除及降主动脉移植置换术。术后三个月的TTE显示左心室收缩功能恢复正常,左心室射血分数为58%,左心室扩张情况改善,肥厚消退,E/e'比值降至19.5。术前左心室收缩功能障碍似乎归因于后负荷不匹配。术后两年,患者健康状况良好,未出现心力衰竭或任何与主动脉相关的事件。
成人主动脉缩窄预后较差,由于慢性后负荷增加会对心脏功能产生影响。我们报告一例老年主动脉缩窄患者,其左心室收缩功能障碍在手术修复后得到改善。我们认为主动脉缩窄的手术修复解决了偏心性肥厚问题,并纠正了术前存在的后负荷不匹配,从而导致左心室收缩功能改善。