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Mechanisms involved in the deposition of immune complexes in tissues.

作者信息

Cochrane C G

出版信息

J Exp Med. 1971 Sep 1;134(3 Pt 2):75s-89s.

PMID:4105809
Abstract
摘要

相似文献

1
Mechanisms involved in the deposition of immune complexes in tissues.免疫复合物在组织中沉积所涉及的机制。
J Exp Med. 1971 Sep 1;134(3 Pt 2):75s-89s.
2
Fate of preformed immune complexes in rabbits and rhesus monkeys.兔和恒河猴体内预先形成的免疫复合物的转归
J Exp Med. 1971 Sep 1;134(3 Pt 2):19s-31s.
3
Acute immune complex disease in rabbits. The role of complement and of a leukocyte-dependent release of vasoactive amines from platelets.兔的急性免疫复合物疾病。补体的作用以及血小板中血管活性胺的白细胞依赖性释放。
J Exp Med. 1971 Mar 1;133(3):554-71. doi: 10.1084/jem.133.3.554.
4
Studies on passive serum sickness. I. The glomerular fine structure of serum sickness nephritis induced by preformed antigen-antibody complexes in the mouse.被动血清病研究。I. 小鼠中预先形成的抗原-抗体复合物诱导的血清病肾炎的肾小球精细结构
Lab Invest. 1971 May;24(5):383-91.
5
Passage of immune complexes through vascular walls. Evidence for the role of an immediate hypersensitivity mechanism and its mediator: platelet-activating factor.免疫复合物通过血管壁。速发型超敏反应机制及其介质——血小板活化因子作用的证据。
Rheumatology. 1975;6:293-302.
6
Antigen quantitation in experimental immune complex glomerulonephritis. I. Acute serum sickness.实验性免疫复合物性肾小球肾炎中的抗原定量。I. 急性血清病
J Immunol. 1970 Aug;105(2):279-90.
7
Immune complex disease. IV. The nature of the circulating complexes associated with glomerulonephritis in the acute BSA-rabbit system.免疫复合物疾病。IV. 急性牛血清白蛋白-兔系统中与肾小球肾炎相关的循环复合物的性质。
Johns Hopkins Med J. 1972 Jun;130(6):335-43.
8
Evidence for the involvement of IgE-basophil system in acute serum sickness.IgE-嗜碱性粒细胞系统参与急性血清病的证据。
Clin Exp Immunol. 1976 Dec;26(3):449-56.
9
Immune-complex disease.免疫复合物病
N Engl J Med. 1969 Apr 17;280(16):878-84. doi: 10.1056/NEJM196904172801610.
10
Platelet involvement in the nephritis of acute serum sickness in rabbits: protection by dipyridamole and FUT-175.血小板在兔急性血清病肾炎中的作用：双嘧达莫和FUT-175的保护作用
Clin Exp Immunol. 1985 Aug;61(2):388-96.

引用本文的文献

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Update on pathogenic mechanisms of systemic necrotizing vasculitis.系统性坏死性血管炎致病机制的最新进展
Curr Rheumatol Rep. 2008 Dec;10(6):430-5. doi: 10.1007/s11926-008-0070-1.
2
Polymorphonuclear leucocytes release a factor(s) that induces platelet aggregation and ATP release after interaction with insoluble and surface-fixed immune complexes.多形核白细胞释放一种因子，该因子在与不溶性及表面固定的免疫复合物相互作用后可诱导血小板聚集和ATP释放。
Clin Exp Immunol. 1982 Sep;49(3):684-94.
3
Immunopathogenesis of the extrahepatic manifestations of hepatitis B virus infection.
乙型肝炎病毒感染肝外表现的免疫发病机制。
Springer Semin Immunopathol. 1981 Apr;3(4):461-72. doi: 10.1007/BF01951493.
4
Presence of complement-fixing anti-endothelial cell antibodies in systemic lupus erythematosus.系统性红斑狼疮中补体结合抗内皮细胞抗体的存在。
J Clin Invest. 1984 Mar;73(3):611-25. doi: 10.1172/JCI111251.
5
Factors influencing immune complex localisation.影响免疫复合物定位的因素。
Rheumatol Int. 1984;4(3):95-109. doi: 10.1007/BF00541177.
6
Thiabendazole-induced suppression of renal damage in a murine model of autoimmune disease.噻苯达唑对自身免疫性疾病小鼠模型肾损伤的抑制作用
Am J Pathol. 1984 May;115(2):204-11.
7
Platelets as target cells in rheumatoid arthritis and systemic lupus erythematosus: a platelet specific immunoglobulin inducing the release reaction.血小板作为类风湿性关节炎和系统性红斑狼疮中的靶细胞：一种诱导释放反应的血小板特异性免疫球蛋白。
Rheumatol Int. 1982;2(2):67-73. doi: 10.1007/BF00541248.
8
Heterophile antibodies and tissue injury. 3. A role for platelets in the development of lethal vascular injury during Forssman shock in guinea pigs.嗜异性抗体与组织损伤。3. 血小板在豚鼠福斯曼休克致死性血管损伤发展中的作用。
Am J Pathol. 1973 Aug;72(2):179-200.
9
[The immunohistology of the glomerulonephritis. (Mediator between pathogenesis and morphology)].肾小球肾炎的免疫组织学。（发病机制与形态学之间的介质）
Klin Wochenschr. 1973 Aug 1;51(15):739-47. doi: 10.1007/BF01468278.
10
The participation of the platelet in the vascular response to endotoxemia in the rabbit eye.血小板在兔眼对内毒素血症的血管反应中的参与情况。
Am J Pathol. 1973 Jan;70(1):25-44.