Vtiurin B V, Kaem R I
Arkh Patol. 1977;39(12):45-51.
Under conditions of a severe burn trauma the experimental P. aeruginosa pneumonia is accompanied by marked destructive changes of all the elements of the hemato-aerial barrier and its increased permeability. At the same time, because of sharp inhibition of the intracellular lysis of bacteria in all elements of the pulmonary reticuloendothelial system, a long-term retention of the pathogenic microflora in the pulmonary tissue is observed. The combination of these factors results in microflora invasion of the blood channel of the lungs, and in this way the lungs acquire an important role as additional portals of entry of infection in severe burn trauma.
在严重烧伤创伤的情况下,实验性铜绿假单胞菌肺炎伴有气血屏障所有成分的明显破坏变化及其通透性增加。同时,由于肺网状内皮系统所有成分中细菌的细胞内溶解受到强烈抑制,观察到致病微生物群在肺组织中长期滞留。这些因素的结合导致微生物群侵入肺部的血道,通过这种方式,肺部在严重烧伤创伤中作为感染的额外入口获得了重要作用。