The response of "obese" (ob-ob) and "diabetic" (db-db) mice to treatments that influence body temperature.

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作者信息

Yen T T, Fuller R W, Pearson D V

出版信息

Comp Biochem Physiol A Comp Physiol. 1974 Oct 1;49(2A):377-85. doi: 10.1016/0300-9629(74)90128-5.

DOI:10.1016/0300-9629(74)90128-5

PMID:4153985

Abstract

摘要

相似文献

The response of "obese" (ob-ob) and "diabetic" (db-db) mice to treatments that influence body temperature.

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Leptin-deficient mice backcrossed to the BALB/cJ genetic background have reduced adiposity, enhanced fertility, normal body temperature, and severe diabetes.回交至BALB/cJ遗传背景的瘦素缺陷型小鼠脂肪量减少、生育力增强、体温正常且患有严重糖尿病。

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Thermogenesis in diabetes-obesity syndromes in mutant mice.

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引用本文的文献

Leptin: Is It Thermogenic?瘦素：它是产热的吗？

Endocr Rev. 2020 Apr 1;41(2):232-60. doi: 10.1210/endrev/bnz016.

SnoRNA Snord116 (Pwcr1/MBII-85) deletion causes growth deficiency and hyperphagia in mice.小核仁RNA Snord116（Pwcr1/MBII-85）缺失导致小鼠生长发育迟缓及摄食过量。

PLoS One. 2008 Mar 5;3(3):e1709. doi: 10.1371/journal.pone.0001709.

Diabetologia. 1980 Aug;19(2):148-53. doi: 10.1007/BF00421862.

Regional blood flow in genetically obese (ob/ob) mice. The importance of brown adipose tissue to the reduced energy expenditure on non-shivering thermogenesis.遗传性肥胖（ob/ob）小鼠的局部血流。棕色脂肪组织对非寒战产热中能量消耗减少的重要性。

Pflugers Arch. 1980 Jun;385(3):193-201. doi: 10.1007/BF00647457.

Diabetes in the mouse due to two mutant genes - a bibliography.由两个突变基因导致的小鼠糖尿病——一份文献目录。

Diabetologia. 1975 Aug;11(4):325-7. doi: 10.1007/BF00422399.

Pflugers Arch. 1979 Jul;380(3):227-32. doi: 10.1007/BF00582901.

An enzymatic defect in the obese (ob/ob) mouse: loss of thyroid-induced sodium- and potassium-dependent adenosinetriphosphatase.肥胖（ob/ob）小鼠的一种酶缺陷：甲状腺诱导的钠钾依赖型三磷酸腺苷酶缺失。

Proc Natl Acad Sci U S A. 1978 Jan;75(1):477-81. doi: 10.1073/pnas.75.1.477.

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