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Acute cerebral oedema during treatment of hyperglycaemia. An experimentsl model.

作者信息

Clements R S, Prockop L D, Winegrad A I

出版信息

Lancet. 1968 Aug 17;2(7564):384-6. doi: 10.1016/s0140-6736(68)90597-7.

DOI:10.1016/s0140-6736(68)90597-7
PMID:4173793
Abstract
摘要

相似文献

1
Acute cerebral oedema during treatment of hyperglycaemia. An experimentsl model.高血糖治疗期间的急性脑水肿。一种实验模型。
Lancet. 1968 Aug 17;2(7564):384-6. doi: 10.1016/s0140-6736(68)90597-7.
2
Hyperglycemia, polyol accumulation, and increased intracranial pressure.高血糖、多元醇蓄积和颅内压升高。
Arch Neurol. 1971 Aug;25(2):126-40. doi: 10.1001/archneur.1971.00490020044005.
3
A mechanism to explain acute cerebral edema during the treatment of diabetic acidosis.一种解释糖尿病酸中毒治疗期间急性脑水肿的机制。
Diabetes. 1968;17:299.
4
Cerebral edema and depression of sensorium in nonketotic hyperosmolar coma.
Diabetes. 1974 Jun;23(6):525-31. doi: 10.2337/diab.23.6.525.
5
Increased cerebrospinal fluid pressure during therapy for diabetic acidosis.糖尿病酸中毒治疗期间脑脊液压力升高。
Trans Assoc Am Physicians. 1971;84:102-12.
6
Polyol metabolism and the development of elevated cerebrospinal fluid pressure following sustained hyperglycemia.多元醇代谢与持续性高血糖后脑脊液压力升高的发展
Neurology. 1968 Mar;18(3):279-80.
7
Increased fructose production by the brain in alloxan diabetes.在四氧嘧啶糖尿病中大脑果糖生成增加。
Brain Res. 1972 Mar 24;38(2):371-5. doi: 10.1016/0006-8993(72)90719-6.
8
Cerebral oedema complicating treated non-ketotic hyperglycaemia.治疗后非酮症高血糖症并发脑水肿。
Lancet. 1969 Aug 16;2(7616):352-3. doi: 10.1016/s0140-6736(69)92702-0.
9
Increased production of fructose by the brain in dogs made diabetic by alloxan and by growth hormone.用四氧嘧啶和生长激素使狗患糖尿病后，其大脑中果糖生成增加。
Diabetes. 1973 Nov;22(11):820-4. doi: 10.2337/diab.22.11.820.
10
Studies on mechanisms of cerebral edema in diabetic comas. Effects of hyperglycemia and rapid lowering of plasma glucose in normal rabbits.糖尿病昏迷时脑水肿机制的研究。高血糖及正常家兔血浆葡萄糖快速降低的影响。
J Clin Invest. 1973 Mar;52(3):571-83. doi: 10.1172/JCI107218.

引用本文的文献

1
Brain injury in children with diabetic ketoacidosis: Review of the literature and a proposed pathophysiologic pathway for the development of cerebral edema.儿童糖尿病酮症酸中毒相关脑损伤：文献综述及脑水肿发生的病理生理学机制探讨。
Pediatr Diabetes. 2021 Mar;22(2):148-160. doi: 10.1111/pedi.13152. Epub 2020 Dec 3.
2
Fructose levels are markedly elevated in cerebrospinal fluid compared to plasma in pregnant women.与孕妇的血浆相比，其脑脊液中的果糖水平显著升高。
PLoS One. 2015 Jun 2;10(6):e0128582. doi: 10.1371/journal.pone.0128582. eCollection 2015.
3
Polyol-pathway enzymes of human brain. Partial purification and properties of sorbitol dehydrogenase.
人类大脑的多元醇途径酶。山梨醇脱氢酶的部分纯化及特性
Biochem J. 1983 Apr 1;211(1):81-90. doi: 10.1042/bj2110081.
4
Localization of aldose reductase and sorbitol dehydrogenase in the nervous system of normal and diabetic rats.正常和糖尿病大鼠神经系统中醛糖还原酶和山梨醇脱氢酶的定位
Acta Diabetol Lat. 1981 Oct-Dec;18(4):373-81. doi: 10.1007/BF02042822.
5
Low-dose insulin treatment of hyperosmolar diabetic coma.低剂量胰岛素治疗高渗性糖尿病昏迷。
Postgrad Med J. 1981 Sep;57(671):556-9. doi: 10.1136/pgmj.57.671.556.
6
[Dialysis-induced hyperglycemic nonketotic coma].[透析诱发的高血糖非酮症昏迷]
Acta Diabetol Lat. 1969 Apr-Jun;6(2):389-94.
7
Hyperosmolar hyperglycaemic non-ketotic diabetic coma complicating open heart surgery.高渗性高血糖非酮症糖尿病昏迷并发心脏直视手术
Br Heart J. 1972 Oct;34(10):1075-7. doi: 10.1136/hrt.34.10.1075.
8
Diabetic ketoacidosis.糖尿病酮症酸中毒
Br Med J. 1970 Oct 24;4(5729):246. doi: 10.1136/bmj.4.5729.246-a.
9
Treatment of severe diabetes mellitus by insulin infusion.胰岛素输注治疗重度糖尿病
Br Med J. 1974 Jun 29;2(5921):691-4. doi: 10.1136/bmj.2.5921.691.
10
Mechanisms of seizures and coma in hypoglycemia. Evidence for a direct effect of insulin on electrolyte transport in brain.低血糖症中癫痫发作和昏迷的机制。胰岛素对脑内电解质转运有直接作用的证据。
J Clin Invest. 1974 Sep;54(3):654-63. doi: 10.1172/JCI107803.