Kramer L D, Cole J P, Messenger J C, Ellestad M H
Chest. 1979 Feb;75(2):189-92. doi: 10.1378/chest.75.2.189.
A 19-year-old white man with familial hypokalemic periodic paralysis developed evidence of cardiac dysfunction during a episode of flaccid paralysis. This consisted of elevated total creatine phosphokinase (CPK), an increased myocardial fraction of CPK (myocardial band), alteration in the lactic dehydrogenase isoenzyme pattern, severe bradycardia, and evidence of left ventricular dysfunction. These findings, in conjunction with selected cases from the literature, suggest the possibility that cardiomyopathy may be a heretofore unrecognized complication of this disorder.
一名患有家族性低钾性周期性麻痹的19岁白人男性在一次弛缓性麻痹发作期间出现了心脏功能障碍的迹象。这包括总肌酸磷酸激酶（CPK）升高、CPK的心肌部分（心肌带）增加、乳酸脱氢酶同工酶模式改变、严重心动过缓以及左心室功能障碍的证据。这些发现，结合文献中的特定病例，提示心肌病可能是这种疾病迄今为止未被认识到的并发症。
