Naeser M A, Hayward R W
Arch Neurol. 1979 Apr;36(4):233-5. doi: 10.1001/archneur.1979.00500400087016.
A 39-year-old man suffered an intracerebral hemorrhage in the region of the left internal capsule deep to Wernicke's area. The location of the lesion was confirmed by computerized tomography (CT) performed two days postictally. Two weeks after admission, the Boston Diagnostic Aphasia Examination (BDAE) diclosed Wernicke's aphasia. We hypothesize that the hematoma exerted pressure on Wernicke's cortical area, thus causing the resulting Wernicke's aphasia at that time. A CT scan three months later showed absorption of the hematoma, with a residual low-density lesion deep to Wernicke's area, in the region of the arcuate fasciculus. At that time, BDAE testing disclosed a mild conduction aphasia. Serial CT scanning combined with discriminating clinical evaluation of aphasia provides a valuable opportunity for study of the processes underlying stroke resolution and aphasia.
一名39岁男性在韦尼克区深部的左侧内囊区域发生脑出血。发病两天后通过计算机断层扫描（CT）确认了病变位置。入院两周后，波士顿失语症诊断检查（BDAE）显示为韦尼克失语症。我们推测血肿对韦尼克皮质区施加了压力，从而导致当时出现韦尼克失语症。三个月后的CT扫描显示血肿吸收，在韦尼克区深部、弓状束区域有残留的低密度病变。当时，BDAE测试显示为轻度传导性失语症。连续CT扫描结合对失语症的鉴别临床评估为研究中风恢复和失语症的潜在过程提供了宝贵机会。
