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Infection of muscle cultures from various species with oncogenic DNA viruses (SV40 and polyoma).

作者信息

Fogel M, Defendi V

出版信息

Proc Natl Acad Sci U S A. 1967 Sep;58(3):967-73. doi: 10.1073/pnas.58.3.967.

DOI:10.1073/pnas.58.3.967
PMID:4293268
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC335733/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/b111d0930de0/pnas00679-0174-f.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/b03afa2bed94/pnas00679-0174-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/01d8be0fe9ee/pnas00679-0174-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/de73d05e2783/pnas00679-0174-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/c0114fc8249a/pnas00679-0174-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/18fcb77b5152/pnas00679-0174-e.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/b111d0930de0/pnas00679-0174-f.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/b03afa2bed94/pnas00679-0174-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/01d8be0fe9ee/pnas00679-0174-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/de73d05e2783/pnas00679-0174-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/c0114fc8249a/pnas00679-0174-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/18fcb77b5152/pnas00679-0174-e.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1440/335733/b111d0930de0/pnas00679-0174-f.jpg

相似文献

1
Infection of muscle cultures from various species with oncogenic DNA viruses (SV40 and polyoma).用致癌DNA病毒（SV40和多瘤病毒）感染来自不同物种的肌肉培养物。
Proc Natl Acad Sci U S A. 1967 Sep;58(3):967-73. doi: 10.1073/pnas.58.3.967.
2
Cell transformation by polyoma virus and SV40.多瘤病毒和猿猴空泡病毒40所致的细胞转化
Nature. 1969 Dec 13;224(5224):1069-71. doi: 10.1038/2241069a0.
3
Histone synthesis in polyoma- and SV40-infected cells.多瘤病毒和SV40感染细胞中的组蛋白合成。
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IMMUNITY IN HAMSTERS TO CELLS TRANSFORMED IN VITRO AND IN VIVO BY SV40. TESTS FOR ANTIGENIC RELATIONSHIP AMONG THE PAPOVAVIRUSES.仓鼠对经SV40体外和体内转化的细胞的免疫性。乳头多瘤空泡病毒之间抗原关系的检测。
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Increase in cellular RNA in cells infected with DNA oncogenic viruses.感染DNA致癌病毒的细胞中细胞RNA增加。
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Neoplastic differentiation: interaction of simian virus 40 and polyoma virus with murine teratocarcinoma cells in vitro.肿瘤分化：猿猴病毒40和多瘤病毒与小鼠畸胎瘤细胞在体外的相互作用。
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Susceptibility to some animal viruses of cells transformed by SV40 or polyoma virus.对由SV40或多瘤病毒转化的细胞对某些动物病毒的易感性。
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[Induction of the "tumor" (T) antigen of SV40 virus in various tissue cultures].[在各种组织培养物中诱导SV40病毒的“肿瘤”（T）抗原]
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本文引用的文献

1
The induction of cellular DNA synthesis by simian virus 40 in contact-inhibited and in x-irradiated cells.猴病毒40在接触抑制细胞和经x射线照射的细胞中诱导细胞DNA合成的研究
Proc Natl Acad Sci U S A. 1966 Sep;56(3):918-25. doi: 10.1073/pnas.56.3.918.
2
THE RELATIONSHIP OF THE CELL CYCLE TO TUMOR GROWTH AND CONTROL OF CELL DIVISION: A REVIEW.细胞周期与肿瘤生长及细胞分裂控制的关系：综述
Cancer Res. 1965 Jun;25:581-95.
3
THE NATURE AND LOCALIZATION OF THE SV 40-INDUCED COMPLEMENT-FIXING ANTIGEN.猴空泡病毒40诱导的补体结合抗原的性质与定位
有缺陷的脱氧核苷三磷酸池会阻碍细胞周期重新激活的终末分化肌肉细胞中的DNA复制。
Cell Death Differ. 2017 May;24(5):774-784. doi: 10.1038/cdd.2017.4. Epub 2017 Feb 10.
4
DNA replication is intrinsically hindered in terminally differentiated myotubes.DNA 复制在终末分化的肌管中受到内在阻碍。
PLoS One. 2010 Jul 13;5(7):e11559. doi: 10.1371/journal.pone.0011559.
5
Reconstitution of cyclin D1-associated kinase activity drives terminally differentiated cells into the cell cycle.细胞周期蛋白D1相关激酶活性的恢复促使终末分化细胞进入细胞周期。
Mol Cell Biol. 2001 Aug;21(16):5631-43. doi: 10.1128/MCB.21.16.5631-5643.2001.
6
p21(CIP1) and p57(KIP2) control muscle differentiation at the myogenin step.p21（CIP1）和p57（KIP2）在肌细胞生成素阶段控制肌肉分化。
Genes Dev. 1999 Jan 15;13(2):213-24. doi: 10.1101/gad.13.2.213.
7
Skeletal muscle cells lacking the retinoblastoma protein display defects in muscle gene expression and accumulate in S and G2 phases of the cell cycle.缺乏视网膜母细胞瘤蛋白的骨骼肌细胞在肌肉基因表达方面表现出缺陷，并在细胞周期的S期和G2期积累。
J Cell Biol. 1996 Oct;135(2):441-56. doi: 10.1083/jcb.135.2.441.
8
Trichinella spiralis infected skeletal muscle cells arrest in G2/M and cease muscle gene expression.旋毛虫感染的骨骼肌细胞停滞在G2/M期并停止肌肉基因表达。
J Cell Biol. 1993 May;121(4):785-93. doi: 10.1083/jcb.121.4.785.
9
SV40 large T antigen-induced inhibition of terminal differentiation of primary skeletal muscle cells is associated with a block in the expression of MyoD and myogenin.SV40大T抗原诱导的原代骨骼肌细胞终末分化抑制与MyoD和肌细胞生成素表达受阻有关。
Mol Biol Rep. 1994;20(3):143-8. doi: 10.1007/BF00990546.
10
Transformation of human skeletal muscle cells by simian virus 40.猿猴病毒40对人骨骼肌细胞的转化
Proc Natl Acad Sci U S A. 1983 Nov;80(21):6581-5. doi: 10.1073/pnas.80.21.6581.
Proc Natl Acad Sci U S A. 1965 Mar;53(3):684-92. doi: 10.1073/pnas.53.3.684.
4
DIFFERENTIAL EFFECTS OF INHIBITORS ON THE STEPS LEADING TO THE FORMATION OF SV40 TUMOR AND VIRUS ANTIGENS.抑制剂对导致SV40肿瘤和病毒抗原形成的各个步骤的不同作用。
J Exp Med. 1965 Jun 1;121(6):935-44. doi: 10.1084/jem.121.6.935.
5
INDUCTION OF CELLULAR DNA SYNTHESIS BY POLYOMA VIRUS.多瘤病毒诱导细胞DNA合成
Proc Natl Acad Sci U S A. 1965 Feb;53(2):403-10. doi: 10.1073/pnas.53.2.403.
6
THE EFFECT OF ACTINOMYCIN D ON HEART AND THIGH MUSCLE CELLS GROWN IN VITRO.放线菌素D对体外培养的心肌细胞和大腿肌肉细胞的作用。
Dev Biol. 1964 Jun;9:347-66. doi: 10.1016/0012-1606(64)90030-2.
7
SOME CHARACTERISTICS OF LARGE-PLAQUE AND SMALL-PLAQUE LINES OF POLYOMA VIRUS.多瘤病毒大斑块和小斑块株系的一些特征
Virology. 1964 Feb;22:235-44. doi: 10.1016/0042-6822(64)90008-x.
8
VIRAL SUSCEPTIBILITY AND EMBRYONIC DIFFERENTIATION. III. CORRELATION BETWEEN AN INDUCTIVE TISSUE INTERACTION AND THE ONSET OF VIRAL RESISTANCE.
J Natl Cancer Inst. 1963 Dec;31:1533-47.
9
SPECIFIC ADENOVIRUS COMPLEMENT-FIXING ANTIGENS IN VIRUS-FREE HAMSTER AND RAT TUMORS.无病毒仓鼠和大鼠肿瘤中的特异性腺病毒补体结合抗原。
Proc Natl Acad Sci U S A. 1963 Aug;50(2):379-89. doi: 10.1073/pnas.50.2.379.
10
Clonal analysis of myogenesis.肌发生的克隆分析。
Science. 1963 Jun 21;140(3573):1273-84. doi: 10.1126/science.140.3573.1273.