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Carcinogenesis by avian sarcoma viruses. X. The decreased requirement for insulin-replaceable activity in serum for cell multiplication.

作者信息

Temin H M

出版信息

Int J Cancer. 1968 Nov 15;3(6):771-87. doi: 10.1002/ijc.2910030611.

DOI:10.1002/ijc.2910030611
PMID:4307758
Abstract
摘要

相似文献

1
Carcinogenesis by avian sarcoma viruses. X. The decreased requirement for insulin-replaceable activity in serum for cell multiplication.禽肉瘤病毒致癌作用。十、细胞增殖时血清中胰岛素可替代活性需求的降低。
Int J Cancer. 1968 Nov 15;3(6):771-87. doi: 10.1002/ijc.2910030611.
2
Studies on carcinogenesis by avian sarcoma viruses. VI. Differential multiplication of uninfected and of converted cells in response to insulin.禽肉瘤病毒致癌作用的研究。VI. 未感染细胞和转化细胞对胰岛素反应的差异增殖。
J Cell Physiol. 1967 Jun;69(3):377-84. doi: 10.1002/jcp.1040690314.
3
Control of cell multiplication in uninfected chicken cells and chicken cells converted by avian sarcoma viruses.
J Cell Physiol. 1969 Aug;74(1):9-16. doi: 10.1002/jcp.1040740103.
4
Studies on carcinogenesis by avian sarcoma viruses. 3. The differential effect of serum and polyanions on multiplication of uninfected and converted cells.
J Natl Cancer Inst. 1966 Aug;37(2):167-75.
5
[Study of the relations between the tumorigenic virus of quail and Rous sarcoma virus].[鹌鹑致瘤病毒与劳斯肉瘤病毒之间关系的研究]
Ann Inst Pasteur (Paris). 1968 Jul;115(1):122-8.
6
Recovery of a new virus from apparently normal chick cells by infection with avian tumor viruses.通过用禽肿瘤病毒感染从看似正常的鸡细胞中分离出一种新病毒。
Proc Natl Acad Sci U S A. 1970 Dec;67(4):1797-803. doi: 10.1073/pnas.67.4.1797.
7
Control by factors in serum of multiplication of uninfected cells and cells infected and converted by avian sarcoma viruses.血清中各因素对未感染细胞以及被禽肉瘤病毒感染并转化的细胞增殖的控制。
Wistar Inst Symp Monogr. 1967;7:103-16.
8
Carcinogenesis by RNA sarcoma viruses. XII. A quantitative study of infection of rat cells in vitro by avian sarcoma viruses.RNA肉瘤病毒致癌作用。十二、禽肉瘤病毒对大鼠细胞体外感染的定量研究。
Virology. 1970 Jan;40(1):118-34. doi: 10.1016/0042-6822(70)90384-3.
9
Studies of carcinogenesis by avian sarcoma viruses. II. Virus-induced increase in hyaluronic acid synthetase in chicken fibroblasts.禽肉瘤病毒致癌作用的研究。II. 病毒诱导鸡成纤维细胞中透明质酸合成酶增加
J Biol Chem. 1966 May 10;241(9):2052-7.
10
DEAE-dextran: enhancement of cellular transformation induced by avian sarcoma viruses.
Virology. 1967 Sep;33(1):175-7. doi: 10.1016/0042-6822(67)90109-2.

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Insulin resistance in vascular endothelial cells promotes intestinal tumour formation.血管内皮细胞中的胰岛素抵抗促进肠道肿瘤形成。
Oncogene. 2017 Aug 31;36(35):4987-4996. doi: 10.1038/onc.2017.107. Epub 2017 May 1.
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Mitosis in vertebrates: the G2/M and M/A transitions and their associated checkpoints.脊椎动物中的有丝分裂:G2/M 和 M/A 转变及其相关的检验点。
Chromosome Res. 2011 Apr;19(3):291-306. doi: 10.1007/s10577-010-9178-z.
3
Exogenous thymidine is preferentially incorporated into human cytomegalovirus DNA in infected human fibroblasts.
外源性胸苷优先掺入受感染的人成纤维细胞中的人巨细胞病毒DNA中。
J Virol. 1996 Sep;70(9):6402-4. doi: 10.1128/JVI.70.9.6402-6404.1996.
4
Analysis of the reduced growth factor dependency of simian virus 40-transformed 3T3 cells.猿猴病毒40转化的3T3细胞对生长因子依赖性降低的分析。
Mol Cell Biol. 1984 Aug;4(8):1572-6. doi: 10.1128/mcb.4.8.1572-1576.1984.
5
Replication of reticuloendotheliosis viruses in cell culture: acute infection.网状内皮组织增殖症病毒在细胞培养中的复制:急性感染
J Virol. 1974 Feb;13(2):291-7. doi: 10.1128/JVI.13.2.291-297.1974.
6
Control of proliferation in two cell lines in vitro.体外两种细胞系增殖的控制
Biophysik. 1972;9(1):70-84. doi: 10.1007/BF01293481.
7
The epidermal cell migration promoting activity of serum in guinea pig skin in vitro.豚鼠皮肤血清促进体外表皮细胞迁移的活性。
Arch Dermatol Forsch. 1974 Jun 11;249(4):367-72. doi: 10.1007/BF00557897.
8
A unifying hypothesis concerning the nature of malignant growth.关于恶性生长本质的一个统一假说。
Proc Natl Acad Sci U S A. 1972 Oct;69(10):2840-1. doi: 10.1073/pnas.69.10.2840.
9
Growth-and density-dependent inhibition of deoxyglucose transport in Balb 3T3 cells and its absence in cells transformed by murine sarcoma virus.Balb 3T3细胞中脱氧葡萄糖转运的生长和密度依赖性抑制及其在鼠肉瘤病毒转化细胞中的缺失。
Proc Natl Acad Sci U S A. 1973 Aug;70(8):2374-8. doi: 10.1073/pnas.70.8.2374.
10
New growth and viruses.新的生长与病毒。
Br Med J. 1970 Sep 5;3(5722):541-5. doi: 10.1136/bmj.3.5722.541.